Activation of CXCL16/CXCR6 axis aggravates cardiac ischemia/reperfusion injury by recruiting the IL-17a-producing CD1d + T cells.

Autor: Wang S; Shanghai Institute of Cardiovascular Diseases, Zhongshan Hospital, Fudan University, Shanghai, China.; Institutes of Biomedical Sciences, Fudan University, Shanghai, China., Ma L; Shanghai Institute of Cardiovascular Diseases, Zhongshan Hospital, Fudan University, Shanghai, China., Yang J; Shanghai Institute of Cardiovascular Diseases, Zhongshan Hospital, Fudan University, Shanghai, China., Dong Z; Shanghai Institute of Cardiovascular Diseases, Zhongshan Hospital, Fudan University, Shanghai, China., Wu J; Shanghai Institute of Cardiovascular Diseases, Zhongshan Hospital, Fudan University, Shanghai, China.; Institutes of Biomedical Sciences, Fudan University, Shanghai, China., Lu X; Department of outpatient, Shanghai Yangpu Hospital of Traditional Chinese Medicine, Shanghai, China., Wang J; Department of Cardiology, Yangpu Hospital, Tongji University School of Medicine, Shanghai, China., Dai F; Shanghai Institute of Cardiovascular Diseases, Zhongshan Hospital, Fudan University, Shanghai, China., Tu G; Shanghai Institute of Cardiovascular Diseases, Zhongshan Hospital, Fudan University, Shanghai, China., Xu L; Shanghai Institute of Cardiovascular Diseases, Zhongshan Hospital, Fudan University, Shanghai, China., Zhao G; Shanghai Institute of Cardiovascular Diseases, Zhongshan Hospital, Fudan University, Shanghai, China., Zhang F; Shanghai Institute of Cardiovascular Diseases, Zhongshan Hospital, Fudan University, Shanghai, China., Zou Y; Shanghai Institute of Cardiovascular Diseases, Zhongshan Hospital, Fudan University, Shanghai, China.; Institutes of Biomedical Sciences, Fudan University, Shanghai, China., Ge J; Shanghai Institute of Cardiovascular Diseases, Zhongshan Hospital, Fudan University, Shanghai, China.; Institutes of Biomedical Sciences, Fudan University, Shanghai, China.
Jazyk: angličtina
Zdroj: Clinical and translational medicine [Clin Transl Med] 2021 Feb; Vol. 11 (2), pp. e301.
DOI: 10.1002/ctm2.301
Databáze: MEDLINE