Cardiac Conduction Defects in Systemic Lupus Erythematosus.
| Autor: | Butt S; Internal Medicine, Baptist Memorial Hospital, Oxford, USA., Kiran S; Pulmonology and Critical Care, University of Cincinnati Medical Center, Cincinnati, USA., Qadir N; Infectious Diseases, University of Louisville, Louisville, USA., Menghani D; Infectious Diseases, University of Louisville, Louisville, USA., Tanzeem H; Medicine, Dow Medical College/Civil Hospital, Karachi, PAK. |
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| Jazyk: | angličtina |
| Zdroj: | Cureus [Cureus] 2020 Oct 10; Vol. 12 (10), pp. e10882. Date of Electronic Publication: 2020 Oct 10. |
| DOI: | 10.7759/cureus.10882 |
| Abstrakt: | Systemic autoimmune conditions may cause morbidity and mortality. Systemic lupus erythematosus (SLE) is a prominent example of such diseases. It can result in conduction abnormalities due to accelerated atherosclerosis, vasculitis, or autoantibodies-induced myocarditis. Cardiac conduction abnormalities may produce sinus tachycardia, sinus bradycardia, prolonged QT intervals, atrial fibrillation, or atrioventricular (AV) nodal blocks. Neonatal lupus is sometimes associated with anti-Ro/SSA and anti-La/SSB antibodies, but their role remains a matter of controversy in adults. Competing Interests: The authors have declared that no competing interests exist. (Copyright © 2020, Butt et al.) |
| Databáze: | MEDLINE |
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