Severe atrial fibrosis as a cause of significant intraatrial conduction delay in a patient with scleromyositis.

Autor: Kaczmarek K; Department of Electrocardiology, Medical University of Lodz, Pomorska Str 251, 92-213 Lodz, Poland., Poddębska I; Department of Electrocardiology, Medical University of Lodz, Pomorska Str 251, 92-213 Lodz, Poland., Kałowski M; Department of Electrocardiology, Medical University of Lodz, Pomorska Str 251, 92-213 Lodz, Poland., Berner R; Department of Electrocardiology, Medical University of Lodz, Pomorska Str 251, 92-213 Lodz, Poland., Zatorska-Berner M; Department of Electrocardiology, Medical University of Lodz, Pomorska Str 251, 92-213 Lodz, Poland., Ptaszyński P; Department of Electrocardiology, Medical University of Lodz, Pomorska Str 251, 92-213 Lodz, Poland., Cygankiewicz I; Department of Electrocardiology, Medical University of Lodz, Pomorska Str 251, 92-213 Lodz, Poland. Electronic address: iwona.cygankiewicz@umed.lodz.pl.
Jazyk: angličtina
Zdroj: Journal of electrocardiology [J Electrocardiol] 2019 Sep - Oct; Vol. 56, pp. 77-80. Date of Electronic Publication: 2019 Jun 19.
DOI: 10.1016/j.jelectrocard.2019.06.012
Abstrakt: Scleromyositis is an autoimmune disease and an overlap syndrome of scleroderma and poly/dermatomyositis. It is characterized by frequent cardiovascular involvement including heart failure, arrhythmias and conduction disturbances. We present a case of a 73-year old female patient who required an upgrade from a DDD pacemaker to cardiac resynchronization therapy due left ventricular dysfunction and permanent ventricular pacing. Electroanatomical mapping (CARTO 3D) revealed extensive right atrial fibrosis which resulted in significant delay in intraatrial conduction. Interval from atrial paced stimulus to A signal in His bundle was 364 ms, while AH and HV intervals were within normal range.
(Copyright © 2019 Elsevier Inc. All rights reserved.)
Databáze: MEDLINE
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