Ultrasound, Clinical, and Electrophysiological Findings in Persistent Carpal Tunnel Syndrome.

Autor: Kerasnoudis A; Department of Neurology, St. Luke's Hospital, Thessaloniki, Greece.; Department of Neurology, St. Josef Hospital, Bochum, Germany., Barmpalios G; Department of Radiology, Iatriki Diagnosi Serron, Serres, Greece., Ntasiou P; Department of Neurology, St. Luke's Hospital, Thessaloniki, Greece., Lakkos T; Department of Orthopedic Surgery, St. Luke's Hospital, Thessaloniki, Greece., Venouziou A; Department of Orthopedic Surgery, St. Luke's Hospital, Thessaloniki, Greece.
Jazyk: angličtina
Zdroj: Journal of neuroimaging : official journal of the American Society of Neuroimaging [J Neuroimaging] 2019 Mar; Vol. 29 (2), pp. 218-222. Date of Electronic Publication: 2018 Nov 23.
DOI: 10.1111/jon.12585
Abstrakt: Background and Purpose: We present the clinical, electrophysiological, and nerve ultrasound findings in cases of persistent carpal tunnel syndrome (PCTS).
Methods: Eighteen PCTS patients underwent evaluation with the Boston Carpal Tunnel Syndrome Questionnaire (BCTSQ), electrophysiology, and nerve ultrasound with a mean of 3.5 months (SD ± 1.4) after open surgery.
Results: PCTS patients showed a mean symptom severity scale score of 3.1 (SD ± 1.1) and functional severity scale score of 3.2 (SD ± 0.9) in BCTSQ. Nerve conduction studies revealed axonal affection of the median nerve in 13/18 patients, ultrasound showed disturbed echogenicity in all patients, a pathological wrist to forearm ratio in 16/18 patients, and cross-sectional area enlargement of the median nerve at the distal wrist crease in 12/18 patients. Ultrasound documented scar tissue formation (in 12/18 patients), incomplete release of retinaculum flexorum (in 4/18 patients), and neuroma of the median nerve (in 2/18 patients) as PCTS cause.
Conclusion: Our data show significant functional disability, axonal nerve damage, and scar tissue formation as common PCTS causes.
(© 2018 by the American Society of Neuroimaging.)
Databáze: MEDLINE
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