| Autor: |
Jayaschandran V; Department of Transplant Program, North Shore University Hospital, New York., Rajendran PP; Department of Transplant Program, North Shore University Hospital, New York., Khanal M; Department of Transplant Program, North Shore University Hospital, New York., Krishnan P; Department of Transplant Program, North Shore University Hospital, New York., Ramadas P; Department of Transplant Program, North Shore University Hospital, New York., Alex A; Department of Transplant Program, North Shore University Hospital, New York., Nambiar L; Department of Transplant Program, North Shore University Hospital, New York., Hirschwerk DA; Department of Infectious Disease Medicine, North Shore University Hospital, New York., McGowan JP; Department of Infectious Disease Medicine, North Shore University Hospital, New York., Ali N; Department of Transplant Program, North Shore University Hospital, New York., Basu A; Department of Transplant Program, North Shore University Hospital, New York., Bhaskaran M; Department of Transplant Program, North Shore University Hospital, New York., Molmenti EP; Department of Transplant Program, North Shore University Hospital, New York. |
| Abstrakt: |
Current research states that AIDS pathogenesis has its roots in a chronic activation of immune system secondary to human immunodeficiency virus (HIV)-induced proliferation of T cells, B cells, NK cells, and macrophages. Immune activation due to acute HIV infection can be highly detrimental to allograft survival in a renal transplant recipient. In this report, we describe a 32-year-old African-American male patient who underwent a second live donor renal transplant, following which he developed acute allograft rejection coincident with newly acquired HIV seropositivity. |
