Left ventricular characteristics during exercise in patients after Fontan's operation for tricuspid atresia
Autor: | Atsuyoshi Takao, Chisato Kondoh, Toshio Nakanishi, Yasuharu Imai, Makoto Nakazawa, Michiaki Hiroe, Seimei Nakae |
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Rok vydání: | 1988 |
Předmět: |
Adult
Male medicine.medical_specialty Supine position Adolescent Heart disease Heart Ventricles Hemodynamics Postoperative Complications Internal medicine medicine Humans cardiovascular diseases Tricuspid atresia Child Ejection fraction Tricuspid valve business.industry Stroke Volume Stroke volume medicine.disease Preload medicine.anatomical_structure Child Preschool Anesthesia Exercise Test cardiovascular system Cardiology Female Tricuspid Valve Cardiology and Cardiovascular Medicine business Follow-Up Studies |
Zdroj: | Heart and Vessels. 4:34-39 |
ISSN: | 1615-2573 0910-8327 |
DOI: | 10.1007/bf02058685 |
Popis: | Left ventricular function during supine bicycle exercise was studied using multigated blood pool imaging in ten patients with tricuspid atresia after Fontan's operation and in 13 children and adults (control group). The mean age of the patients was 16 years and the mean interval between operation and study was 5 years. The peak work loads that the patients could perform were similar to those in the control group. Work loads and heart rates during radionuclide study in the operated group were also similar to those in the control group. The left ventricular ejection fraction at rest and during exercise in the operated group was less than in the control group, although the net increase during exercise was similar in the two groups. During exercise, left ventricular end-diastolic volume decreased significantly in the operated group. In the control group, this variable did not change significantly. Left ventricular stroke volume increased during exercise in the control group but it did not change significantly in the operated group. These data indicate that in patients after Fontan's operation, left ventricular performance remains low during exercise, which is in part due to diminished left ventricular preload reserve, and this in turn may be caused by reduced reserve of right heart output. |
Databáze: | OpenAIRE |
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