Abnormal molecular response to Takayasu arteritis causing extensive large-vessel calcification.
| Autor: | Garland BT; Division of Vascular Surgery, University of Washington, Seattle., Boehm M; Center for Molecular Medicine, National Institutes of Health, Bethesda., Grayson PC; Center for Molecular Medicine, National Institutes of Health, Bethesda., Hilaire CS; Center for Molecular Medicine, National Institutes of Health, Bethesda., Brofferio A; Center for Molecular Medicine, National Institutes of Health, Bethesda., Starnes BW; Division of Vascular Surgery, University of Washington, Seattle. |
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| Jazyk: | angličtina |
| Zdroj: | Journal of vascular surgery cases and innovative techniques [J Vasc Surg Cases Innov Tech] 2016 Dec; Vol. 2 (4), pp. 190-192. |
| DOI: | 10.1016/j.jvscit.2016.05.004 |
| Abstrakt: | Takayasu arteritis is a large-vessel vasculitis that often results in pulselessness due to fibrotic stenoses. Whereas minor calcification is sometimes seen with Takayasu arteritis, it rarely causes stenosis. Extensive calcification resulting in malperfusion is exceedingly rare and has been attributed to disorders in calcium trafficking in a chronic inflammatory state. We report an unusual case of rapidly progressive and extensive aortic calcification in the setting of Takayasu arteritis. Competing Interests: Author conflict of interest: none. The editors and reviewers of this article have no relevant financial relationships to disclose per the Journal policy that requires reviewers to decline review of any manuscript for which they may have a conflict of interest. |
| Databáze: | MEDLINE |
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