Zobrazeno 1 - 10
of 127
pro vyhledávání: '"tumor necrosis factor receptor-associated factors"'
Publikováno v:
European Journal of Medical Research, Vol 29, Iss 1, Pp 1-18 (2024)
Abstract Background Tumor necrosis factor receptor-associated factors family genes play a pivotal role in tumorigenesis and metastasis, functioning as adapters or E3 ubiquitin ligases across various signaling pathways. To date, limited research has e
Externí odkaz:
https://doaj.org/article/f9b05df82ffd45bc91e2872f2e21795a
Akademický článek
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Akademický článek
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Publikováno v:
Frontiers in Pharmacology, Vol 10 (2019)
Background: Increasing evidence suggests that Fbxo3 signaling has an important impact on the pathophysiology of the inflammatory process. Fbxo3 protein inhibition has reduced cytokine-driven inflammation and improved disease severity in animal model
Externí odkaz:
https://doaj.org/article/2559dabb552549648608d2c20dbb0361
Autor:
Takanori So
Publikováno v:
International Immunology. 34:7-20
The tumor necrosis factor receptor (TNFR)-associated factor (TRAF) family of molecules are intracellular signaling adaptors and control diverse signaling pathways mediated not only by the TNFR superfamily and the Toll-like receptor/IL-1 receptor supe
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::13f57f62847e6ceb6f8e1c4c1a7f5487
https://doi.org/10.1093/intimm/dxab058
https://doi.org/10.1093/intimm/dxab058
Autor:
Suzanne A. B. M. Aarts, Tom T. P. Seijkens, Koos J. F. van Dorst, Christine D. Dijkstra, Gijs Kooij, Esther Lutgens
Publikováno v:
Frontiers in Immunology, Vol 8 (2017)
The CD40–CD40L dyad is an immune checkpoint regulator that promotes both innate and adaptive immune responses and has therefore an essential role in the development of inflammatory diseases, including multiple sclerosis (MS). In MS, CD40 and CD40L
Externí odkaz:
https://doaj.org/article/66194d9db3fe45208049f43ccb2d202f
Publikováno v:
Journal of integrative plant biologyREFERENCES. 64(2)
Tumor necrosis factor receptor-associated factor (TRAF) proteins are conserved in higher eukaryotes and play key roles in transducing cellular signals across different organelles. They are characterized by their C-terminal region (TRAF-C domain) cont
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4794be3874f91a9825e8722096f25823
https://pubmed.ncbi.nlm.nih.gov/34676666
https://pubmed.ncbi.nlm.nih.gov/34676666
Publikováno v:
International Journal of Molecular Sciences
Volume 22
Issue 11
International Journal of Molecular Sciences, Vol 22, Iss 6136, p 6136 (2021)
Volume 22
Issue 11
International Journal of Molecular Sciences, Vol 22, Iss 6136, p 6136 (2021)
Tumor necrosis factor receptor-associated factors (TRAFs) play important roles in the biological processes of immune regulation, the inflammatory response, and apoptosis. TRAF4 belongs to the TRAF family and plays a major role in many biological proc
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::042cc3e2c7d6e27fafcc4aeb2cd5d1c6
https://pubmed.ncbi.nlm.nih.gov/34200212
https://pubmed.ncbi.nlm.nih.gov/34200212
Publikováno v:
Fishshellfish immunology. 115
Tumor necrosis factor receptor-associated factor 5 (TRAF5) is an intracellular protein that binds to the cytoplasmic portion of tumor necrosis factor receptors and mediates the activation of downstream nuclear factor-kappa B (NF-κB), interferon regu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9d6587062d69bcdcc74fed6189a35171
https://pubmed.ncbi.nlm.nih.gov/34062236
https://pubmed.ncbi.nlm.nih.gov/34062236
Publikováno v:
Integr Biol (Camb)
The nuclear factor kappa-light-chain-enhancer of activated B cells (NF-κB) is one of the most important transcription factors involved in the regulation of inflammatory signaling pathways. Inappropriate activation of these pathways has been linked t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fa470833007bc6ebd90b2412af8c9bfd
https://pubmed.ncbi.nlm.nih.gov/33893499
https://pubmed.ncbi.nlm.nih.gov/33893499