Zobrazeno 1 - 10
of 10
pro vyhledávání: '"tnfsf4 gene"'
Autor:
Alshaymaa A Ibrahim, Maha Abdelhadi, Ingy Ashmawy, Abeer Ramadan, Aliaa Wahby, Mirhane Hassan, Fatema T Elgengehy, Noha M Abdel Baki
Publikováno v:
Journal of the Arab Society for Medical Research, Vol 18, Iss 1, Pp 76-81 (2023)
Background/aim The disclosure of new gene polymorphisms and their association with rheumatoid arthritis (RA) susceptibility open new windows for better clarification of disease pathogenesis, leading to discovering new therapeutic targets. The present
Externí odkaz:
https://doaj.org/article/a051ed101fb24e308d689364dfa8a215
Background: Tumor necrosis factor superfamily member 4 (TNFSF4) has significant role in modulating autoimmune diseases (ADs) and single nucleotide polymorphism (SNP) is also related with the susceptibility to some diseases. So a meta-analysis aimed a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5ea93964a502f4ab7657b61480018f86
Publikováno v:
Anatolian Journal of Cardiology
Objective: To evaluate the association between the tumor necrosis factor superfamily member 4 (TNFSF4) gene polymorphisms and common cardiovascular and cerebrovascular diseases. Methods: A literature-based search was conducted through databases inclu
Publikováno v:
Computational Biology and Bioinformatics. 7:5
Tumor Necrosis Factor Ligand Superfamily Member4 (TNFSF4) has a huge family of physically homologous transmembrane proteins that regulate various functions in responding cells containing proliferation, differentiation, apoptosis, and inflammatory gen
Publikováno v:
Experimental and Therapeutic Medicine. 2:275-280
Coronary heart disease (CHD) is a complex disorder resulting from the interaction of a number of genetic and environmental factors. Increasing evidence has shown that OX40 ligand (OX40L), also known as tumor necrosis factor superfamily member 4 (TNFS
Autor:
Xuejun Zhang, Sheng Quan Zhang, Wen Sheng Lu, Sen Yang, Liangdan Sun, Xianyong Yin, Jian Wen Han
Publikováno v:
Rheumatology International. 31:227-231
Systemic lupus erythematosus (SLE) is a prototypic autoimmune disease with complex genetic inheritance. Recently, single-nucleotide polymorphisms (SNPs) in tumor necrosis factor (TNF) superfamily gene TNFSF4 have been shown to be associated with SLE
Autor:
Filemon K. Tan, Gene Paz, Shervin Assassi, Frank C. Arnett, Hilda T. Draeger, John D. Reveille, Pravitt Gourh, Dipal Divecha, Sandeep K. Agarwal, Terry A. McNearney, Maureen D. Mayes
Publikováno v:
Annals of the Rheumatic Diseases
ObjectiveIt is increasingly being appreciated that multiple autoimmune diseases share common susceptibility genes. The tumour necrosis factor ligand superfamily member 4 gene (TNFSF4, OX40L), which encodes for the T cell costimulatory molecule OX40 l
Autor:
Susanna Boquist, Jacob Lagercrantz, Massimiliano Ria, Ann Samnegård, Per Eriksson, Anders Hamsten
Publikováno v:
PLoS ONE, Vol 6, Iss 3, p e17652 (2011)
PLoS ONE
PLoS ONE
Background The TNFSF4/TNFRSF4 system, along with several other receptor-ligand pairs, is involved in the recruitment and activation of T-cells and is therefore tentatively implicated in atherosclerosis and acute coronary syndromes. We have previously
Autor:
Marika Kvarnström, Johan G. Brun, Lars Rönnblom, Peter Eriksson, Christopher Sjöwall, Lilian Vasaitis, Gunnel Nordmark, Marie Wahren-Herlenius, Maija-Leena Eloranta, Peter Söderkvist, Svein Joar Auglænd Johnsen, Anders Lundmark, Silke Appel, Malin V. Jonsson, Per Lundmark, Roald Omdal, Elke Theander, Ann-Christine Syvänen, Nicolas Delaleu, Gudlaug Kristjansdottir, Gunnar V. Alm, Eva Baecklund, Roland Jonsson, Erna Harboe, Lasse G. Gøransson
Publikováno v:
Genes and immunity. 12(2)
We performed a candidate gene association study in 540 patients with primary Sjogren's Syndrome (SS) from Sweden (n = 344) and Norway (n = 196) and 532 controls (n = 319 Swedish, n = 213 Norwegian). A total of 1139 single-nucleotide polymorphisms (SN
Autor:
Patricia Carreira, Nicolas Hunzelmann, Jca Broen, Blanca Rueda, P. García de la Peña, Annemie J. Schuerwegh, Javier Martin, Jane Worthington, Carmen P. Simeon, Annika Nordin, Mjh Coenen, Ariane L. Herrick, Norberto Ortego-Centeno, Torsten Witte, Paul G. Shiels, Roger Hesselstrand, Lorenzo Beretta, María Victoria Egurbide, Alexandre E. Voskuyl, Esther Vicente-Rabaneda, J.M. van Laar, Nuria Navarrete, Øyvind Palm, E. De Langhe, M. T. Camps, Alexander Kreuter, Kathleen Claes, Vanessa Smith, Christopher P. Denton, Miguel A. Gonzalez-Gay, Carmen Fonseca, Rajan Madhok, Benedicte A. Lie, I. Gómez-Gracia, Ivan Castellví, Trdj Radstake, Bobby P. C. Koeleman, Meng May Chee, María Cruz Gallego, F De Keyser, V Follonosa, M F González-Escribano, Luis Rodriguez Rodriguez, Lara Bossini-Castillo, Frédéric Houssiau, Leonid Padykov, José Andrés Román-Ivorra, Gabriela Riemekasten, Madelon C. Vonk, A. Pros, Antonio Fernández-Nebro, Paolo Airò, C. Tolosa, Francisco J. García-Hernández, Rene Westhovens, Gerard Espinosa, Raffaella Scorza
Publikováno v:
Europe PubMed Central
Journal of Translational Medicine, Vol 8, Iss Suppl 1, p P5 (2010)
Digital.CSIC. Repositorio Institucional del CSIC
instname
Journal of Translational Medicine
Journal of Translational Medicine, Vol 8, Iss Suppl 1, p P5 (2010)
Digital.CSIC. Repositorio Institucional del CSIC
instname
Journal of Translational Medicine
2 páginas.-- Póster presentado al 5º European Workshop on Immune-Mediated Inflammatory Diseases celebrado en Sitges (Barcelona, España) del 1 al 3 de Diciembre de 2010.-- et al.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c34d27fd640e9c649125a8759da38b16
http://europepmc.org/abstract/PMC/PMC3007795
http://europepmc.org/abstract/PMC/PMC3007795