Zobrazeno 1 - 4
of 4
pro vyhledávání: '"tian-fu Yu"'
Autor:
Ze-qing Pu, Tian-fu Yu, Dong Liu, Cheng-wen Jin, Esha Sadiq, Xiaofei Qiao, Xiaojie Li, Yuxuan Chen, Jinsong Zhang, Mingzhong Tian, Siying Li, Ru-xing Zhao, Xiang-dong Wang
Publikováno v:
Cell Death Discovery, Vol 7, Iss 1, Pp 1-13 (2021)
Abstract Under adverse conditions, such as sustained or chronic hyperglycemia or hyperlipidemia, ROS (reactive oxygen species) or/and ER-stress (endoplasmic reticulum stress) will be induced in pancreatic β cells. ROS or ER-stress damages β-cells e
Externí odkaz:
https://doaj.org/article/9782f4b6ceb7453086a740ff701d3c2b
Autor:
Dong Liu, Yuantao Liu, Chen Zong, Tian‐fu Yu, Esha Sadiq, Jie Cheng, Ze‐qing Pu, Yuchao Zhang, Xiangdong Wang, Xiao Yu, Ruxing Zhao, Cheng‐wen Jin, Dandan Qin, Xia Li, Jicui Chen, Hanse Pablick Patherny Lobo Mouguegue, Jing-Yu Lin, Qingbo Guan
Publikováno v:
Journal of Cellular and Molecular Medicine
Sustained hyperglycaemia and hyperlipidaemia incur endoplasmic reticulum stress (ER stress) and reactive oxygen species (ROS) overproduction in pancreatic β‐cells. ER stress or ROS causes c‐Jun N‐terminal kinase (JNK) activation, and the activ
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d3c998067b9331388ff92e5634b301b9
https://doi.org/10.1111/jcmm.16028
https://doi.org/10.1111/jcmm.16028
Autor:
Mingzhong Tian, Xiaojie Li, Cheng‐wen Jin, Siying Li, Tian‐fu Yu, Jinsong Zhang, Ze‐qing Pu, Dong Liu, Xiaofei Qiao, Esha Sadiq, Ruxing Zhao, Yuxuan Chen, Xiangdong Wang
Publikováno v:
Cell Death Discovery, Vol 7, Iss 1, Pp 1-13 (2021)
Cell Death Discovery
Cell Death Discovery
Under adverse conditions, such as sustained or chronic hyperglycemia or hyperlipidemia, ROS (reactive oxygen species) or/and ER-stress (endoplasmic reticulum stress) will be induced in pancreatic β cells. ROS or ER-stress damages β-cells even leads
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7d9044fae3da81663f788bb2de1d111f
https://doi.org/10.1038/s41420-021-00521-0
https://doi.org/10.1038/s41420-021-00521-0