Zobrazeno 1 - 4
of 4
pro vyhledávání: '"rd Robert T. Davis"'
Autor:
R. John Solaro, David F. Wieczorek, Minae Kobayashi, Michelle M. Monasky, rd Robert T. Davis, Shamim A. K. Chowdhury, Tomoyoshi Kobayashi, Beata M. Wolska, Sumeyye Yar, Sudarsan Rajan, Vadim Gaponenko
Publikováno v:
Journal of Biological Chemistry. 288:16235-16246
α-Tropomyosin (α-TM) has a conserved, charged Asp-137 residue located in the hydrophobic core of its coiled-coil structure, which is unusual in that the residue is found at a position typically occupied by a hydrophobic residue. Asp-137 is thought
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ea8aaf73eca3ed448b7e9b059eb02c3e
https://doi.org/10.1074/jbc.m113.458695
https://doi.org/10.1074/jbc.m113.458695
Autor:
rd Robert T. Davis, John N. Stabley, Bradley J. Behnke, Michael W. Ramsey, Danielle J. McCullough, Judy M. Muller-Delp, nd James M. Dominguez, Michael D. Delp
Publikováno v:
Journal of Applied Physiology. 113:1699-1708
With old age, blood flow to the high-oxidative red skeletal muscle is reduced and blood flow to the low-oxidative white muscle is elevated during exercise. Changes in the number of feed arteries perforating the muscle are thought to contribute to thi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ca48ca775528260432f92e320bb97471
https://doi.org/10.1152/japplphysiol.01025.2012
https://doi.org/10.1152/japplphysiol.01025.2012
Autor:
David M. Ryba, R. John Solaro, David F. Wieczorek, rd Robert T. Davis, Paul T. Mungai, Beata M. Wolska, Madhusudhan Tarigopula, Conrad L. Cowan, Jonathan D. Violin
Publikováno v:
Cardiovascular research. 107(2)
Aims Therapeutic approaches to treat familial dilated cardiomyopathy (DCM), which is characterized by depressed sarcomeric tension and susceptibility to Ca2+-related arrhythmias, have been generally unsuccessful. Our objective in the present work was
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5ba7367057c1145703ee6616610c1024
https://pubmed.ncbi.nlm.nih.gov/26045475
https://pubmed.ncbi.nlm.nih.gov/26045475
Autor:
Aaron C. Hinken, Chad M. Warren, R. John Solaro, Fernando Augusto Lavezzo Dias, David F. Wieczorek, Megan S. Utter, Brandon J. Biesiadecki, Eric M. Montminy, Sakthivel Sadayappan, Beata M. Wolska, Jillian N. Simon, Robert D. Gaffin, Marco S.L. Alves, rd Robert T. Davis, Jeffrey Robbins
Publikováno v:
Circulation. Cardiovascular genetics. 7(2)
Background— Hypertrophic cardiomyopathy (HCM) is a common genetic disorder caused mainly by mutations in sarcomeric proteins and is characterized by maladaptive myocardial hypertrophy, diastolic heart failure, increased myofilament Ca 2+ sensitivit
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bf59e3aa0ae082292113101997c8aca9
https://pubmed.ncbi.nlm.nih.gov/24585742
https://pubmed.ncbi.nlm.nih.gov/24585742