Zobrazeno 1 - 9
of 9
pro vyhledávání: '"payam siyadat"'
Autor:
Maryam Sheikhi, Payam Siyadat, Mehrdad Rostami, Mohammad Hadi Sadeghian, Elnaz Zahiri, Mohammad Ghorbani, Hossein Ayatollahi, Amirali Ayatollahi, Reza Hemmatan Attarbashi, Zahra Khoshnegah
Publikováno v:
Caspian Journal of Internal Medicine, Vol 15, Iss 4, Pp 579-588 (2024)
Background: NUP98 gene fusions in acute myeloid leukemia (AML) have recently attracted much interest. Despite substantial research illuminating the roles of NUP98 fusions in the course of AML, their impacts on the outcome of patients with AML should
Externí odkaz:
https://doaj.org/article/37c5f1a3f0534f11b27842a30bbefa43
Autor:
Maryam Sheikhi, Mehrdad Rostami, Gordon Ferns, Hossein Ayatollahi, Payam Siyadat, Yasamin Ayatollahi, Zahra Khoshnegah
Publikováno v:
Caspian Journal of Internal Medicine, Vol 15, Iss 2, Pp 202-214 (2024)
Background: Although genetic mutations in additional sex-combs-like 1 (ASXL1) are prevalent in acute myeloid leukemia (AML), their exact impact on the AML prognosis remains uncertain. Hence, the present article was carried out to explore the prognost
Externí odkaz:
https://doaj.org/article/014a5c9bdaa040398da4eaaa0b68f249
Autor:
Hossein Ayatollahi, Samaneh Boroumand-Noughabi, Gordon Ferns, Maryam sheikhi, Payam Siyadat, Mehrdad Rostami, zahra khoshnegah
Publikováno v:
Caspian Journal of Internal Medicine, Vol 14, Iss 4, Pp 694-702 (2023)
Background: Autophagy is a pathway for the degradation of cytoplasmic components, which plays an essential role in various cellular and physiological processes, including cell renewal and survival, and immune responses. While recent studies have show
Externí odkaz:
https://doaj.org/article/d83dda836f9946909fb8909383d294fe
Autor:
Sanaz Homayounfar, Hossein Ayatollahi, Gordon Ferns, Reza Hemmatan Attarbashi, Masoumeh Gharib, Maryam Sheikhi, Zahra Khoshnegah, Payam Siyadat, Amirhossein Jafarian
Publikováno v:
Caspian Journal of Internal Medicine, Vol 16, Iss 1, Pp 66-72 (2025)
Background: Comprehensive molecular assessment of cancers could open up new horizons for novel therapies. Fibroblast growth factor receptor 1 (FGFR1) gene amplification has been previously demonstrated in non-small cell lung cancer (NSCLC) patients.
Externí odkaz:
https://doaj.org/article/f5030bbb2d50414ca200ec89abdafcb2
The Emerging Roles of Aldehyde Dehydrogenase in Acute Myeloid Leukemia and Its Therapeutic Potential
Autor:
Hossein Ayatollahi, Atefe Rahmati, Sajad Goudarzi, Maryam Sheikhi, Payam Siyadat, Gordon A Ferns
Publikováno v:
Anti-Cancer Agents in Medicinal Chemistry. 23:246-255
Abstract: Acute myeloid leukemia (AML) is a malignant disorder characterized by myeloid differentiation arrest and uncontrolled clonal expansion of abnormal myeloid progenitor cells. AML is the most common malignant bone marrow (BM) disease in adults
Autor:
ehsan yazdandoust, mohammad hadidi sadeghian, seyyede fatemeh shams, Yasaman Saadatpour, payam siyadat, maryam sheikhi, Monavar Afzal Aghaee, Hossein Ayatollahi
Publikováno v:
Iranian Journal of Pathology. 17:419-426
Autor:
Ehsan, Yazdandoust, Mohammad Hadi, Sadeghian, Seyyede Fatemeh, Shams, Hossein, Ayatollahi, Yasaman, Saadatpour, Payam, Siyadat, Maryam, Sheikhi, Monavvar, Afzalaghaee
Publikováno v:
Iranian journal of pathology. 17(4)
Acute myeloid leukemia (AML) is a hematopoietic malignancy caused by genetic abnormalities. Currently, molecular and genetic factors are routinely used as diagnostic and prognostic markers. FLT-3 is one of the most known diagnostic factors in AML.For
Autor:
Hossein Ayatollahi, Omolbanin Sargazi-Aval, Ali Fani, Payam Siyadat, Maryam Sheikhi, Mohammad Hadi Sadeghian, Ali Bazi
Publikováno v:
Iranian Journal of Pathology
Background & Objective: Acute promyelocytic leukemia (APL) with t(15;17)(q22;q12) is a relatively common subtype of acute myeloid leukemia (AML). Here, our objective was to ascertain the survival of patients with this leukemia in north-east of Iran.
Publikováno v:
Rep Biochem Mol Biol
Scopus-Elsevier
Scopus-Elsevier
BACKGROUND: Acute lymphoblastic leukemia (ALL) is a highly heterogeneous malignancy that accounts for nearly 75% of leukemias in children. While the exact mechanism of ALL is not fully understood, some genetic variants have been implicated as associa
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4cda36127f42215a9dc3f4a701e000a3
https://europepmc.org/articles/PMC8068442/
https://europepmc.org/articles/PMC8068442/