Zobrazeno 1 - 2
of 2
pro vyhledávání: '"pathology [Learning Disabilities]"'
Autor:
Christoph Harms, Nafisa M. Jadavji, Marco Foddis, Ulrich Dirnagl, Janet Lips, Philipp Boehm-Sturm, Ahmed A. Khalil, Martina Füchtemeier, Tracy D. Farr
Publikováno v:
Behavioural brain research 283, 215-226 (2015). doi:10.1016/j.bbr.2015.01.040
Dietary deficiencies in folic acid result in elevated levels of plasma homocysteine, which has been associated with the development of dementia and other neurodegenerative disorders. Previously, we have shown that elevated levels of plasma homocystei
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1a53da5e5acbfbaaf8120dbd2ad3a7c5
https://doi.org/10.1016/j.bbr.2015.01.040
https://doi.org/10.1016/j.bbr.2015.01.040
Autor:
Antonino Natalello, Marco Rasile, Eliana Lauranzano, Sonia Mazzitelli, Davide Pozzi, Isabella Barajon, Matteo Tamborini, Chiara Starvaggi-Cucuzza, Fabia Filipello, Michela Matteoli, Toni Giorgino
Publikováno v:
Acta Neuropathologica Communications 4(1), 110 (2016). doi:10.1186/s40478-016-0381-9
Acta Neuropathologica Communications
Acta Neuropathologica Communications 4 (2016): 110. doi:10.1186/s40478-016-0381-9
info:cnr-pdr/source/autori:Mazzitelli S.; Filipello F.; Rasile M.; Lauranzano E.; Starvaggi-Cucuzza C.; Tamborini M.; Pozzi D.; Barajon I.; Giorgino T.; Natalello A.; Matteoli M./titolo:Amyloid-beta 1-24 C-terminal truncated fragment promotes amyloid-beta 1-42 aggregate formation in the healthy brain/doi:10.1186%2Fs40478-016-0381-9/rivista:Acta Neuropathologica Communications/anno:2016/pagina_da:110/pagina_a:/intervallo_pagine:110/volume:4
Scopus-Elsevier
Acta Neuropathologica Communications
Acta Neuropathologica Communications 4 (2016): 110. doi:10.1186/s40478-016-0381-9
info:cnr-pdr/source/autori:Mazzitelli S.; Filipello F.; Rasile M.; Lauranzano E.; Starvaggi-Cucuzza C.; Tamborini M.; Pozzi D.; Barajon I.; Giorgino T.; Natalello A.; Matteoli M./titolo:Amyloid-beta 1-24 C-terminal truncated fragment promotes amyloid-beta 1-42 aggregate formation in the healthy brain/doi:10.1186%2Fs40478-016-0381-9/rivista:Acta Neuropathologica Communications/anno:2016/pagina_da:110/pagina_a:/intervallo_pagine:110/volume:4
Scopus-Elsevier
Substantial data indicate that amyloid-? (A?), the major component of senile plaques, plays a central role in Alzheimer's Disease and indeed the assembly of naturally occurring amyloid peptides into cytotoxic aggregates is linked to the disease patho
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d80d64826414d967cfe3253aaab12eab
https://doi.org/10.1186/s40478-016-0381-9
https://doi.org/10.1186/s40478-016-0381-9