Zobrazeno 1 - 10
of 2 129
pro vyhledávání: '"ox‐LDL"'
Publikováno v:
Open Medicine, Vol 19, Iss 1, Pp 1376-414 (2024)
The malfunction of endothelial progenitor cells (EPCs) due to ox-LDL is a risk contributor for arteriosclerotic disease. Meanwhile, lycopene possesses anti-inflammatory and antioxidative qualities. This investigation aimed to determine if lycopene ca
Externí odkaz:
https://doaj.org/article/c3407c5ab5b94d93972a95be8c0fc007
Autor:
Jingjing Guo, Laijing Du
Publikováno v:
Frontiers in Cell and Developmental Biology, Vol 12 (2024)
Excess cholesterol accumulation induces the accumulation of foam cells, eventually accelerating atherosclerosis progress. Historically, the mechanisms of macrophage-derived foam cells have attracted attention because of their central role in plaque d
Externí odkaz:
https://doaj.org/article/3a60fdfc08c64305834a54d8939e55d9
Publikováno v:
PeerJ, Vol 12, p e18135 (2024)
Background This study aims to identify a novel gene signature for coronary artery disease (CAD), explore the role of immune cell infiltration in CAD pathogenesis, and assess the cell function of mast cell-expressed membrane protein 1 (MCEMP1) in huma
Externí odkaz:
https://doaj.org/article/17c286fd23554de3abacffbffe2ede78
Publikováno v:
BMC Cardiovascular Disorders, Vol 24, Iss 1, Pp 1-11 (2024)
Abstract LY86, also known as MD1, has been implicated in various pathophysiological processes including inflammation, obesity, insulin resistance, and immunoregulation. However, the role of LY86 in cholesterol metabolism remains incompletely understo
Externí odkaz:
https://doaj.org/article/238caca5fa3046b7ae2a0a6fb20db967
Publikováno v:
BMC Cardiovascular Disorders, Vol 24, Iss 1, Pp 1-11 (2024)
Abstract Background Oxidized low-density lipoprotein (ox-LDL) can initiate and affect almost all atherosclerotic events including endothelial dysfunction. In this text, the role and underlying molecular basis of procyanidin B2 (PCB2) with potential a
Externí odkaz:
https://doaj.org/article/a5ca32df1c3a4a579f945ce017dbbe9b
Publikováno v:
Journal of Inflammation Research, Vol Volume 17, Pp 641-653 (2024)
Jia-Huan He,1,* Xiang-Jun Li,2,* Shi-Peng Wang,1 Xia Guo,1 Hao-Xuan Chu,1 Han-Chi Xu,1 Yu-Shi Wang1 1Department of Cardiology, The First Hospital of Jilin University, Changchun, 13000, People’s Republic of China; 2Department of Experimental
Externí odkaz:
https://doaj.org/article/a03a1b8bedf3409ab5d5377da5e70981
Publikováno v:
Communication in Biomathematical Sciences, Vol 6, Iss 2, Pp 108-125 (2023)
The role of anti-cytokines in atherosclerosis is to reduce inflammation in the intima. In some situations, certain anti-inflammatory cytokines like TGF-beta and IL-6 have shown the characteristics like a pro-inflammatory cytokines, which are showing
Externí odkaz:
https://doaj.org/article/f241f121c8c5494eac346ac7bcc1d04e
Autor:
Jianjie Rong, Chuanyong Li, Qiang Zhang, Guangfeng Zheng, Weijian Fan, Zhichang Pan, Shuming Shi
Publikováno v:
Pharmaceutical Biology, Vol 61, Iss 1, Pp 404-415 (2023)
AbstractContext Ferroptosis may play an essential role in lipid peroxidation and endothelial dysfunction of aortic endothelial cells (ECs) in type 2 diabetes mellitus (T2DM) with atherosclerosis (AS). Hydroxysafflor yellow A (HSYA) has shown substant
Externí odkaz:
https://doaj.org/article/b11451fede5b4e5eb8d82d271345b53a
Publikováno v:
Heliyon, Vol 10, Iss 7, Pp e28887- (2024)
Background: The impairment of endothelial cells triggered by oxidized low-density lipoprotein (ox-LDL) stands as a critical event in the advancement of atherosclerosis (AS). MiR-497-5p has been recognized as a potential predictor for AS, but its prec
Externí odkaz:
https://doaj.org/article/11ac48d2640b4763b1d978213003a8d7
Autor:
Changyi Zhang, Xiaojun Huang, Bin Xie, Danchun Lian, Jinhao Chen, Weiwen Li, Ying Lin, Xiangna Cai, Jilin Li
Publikováno v:
Biomedicine & Pharmacotherapy, Vol 172, Iss , Pp 116268- (2024)
Atherosclerosis is a lipid-driven inflammatory arterial disease, with one crucial factor is oxidized low-density lipoprotein (ox-LDL), which can induce endothelial dysfunction through endoplasmic reticulum stress (ERS). Interleukin-37 (IL-37) exerts
Externí odkaz:
https://doaj.org/article/97127e0ae1e6419f8b65fd158257b1f1