Zobrazeno 1 - 8
of 8
pro vyhledávání: '"methylpiperidinopyrazole"'
Publikováno v:
Molecules, Vol 25, Iss 12, p 2876 (2020)
An estrogen deficiency is the main cause of osteoporosis in postmenopausal women. In bone remodeling, estrogen receptors (ERs) can mediate estrogen-transducing signals. Methylpiperidinopyrazole (MPP) is a highly specific antagonist of ER-alpha (ERα)
Externí odkaz:
https://doaj.org/article/9ab94940c61141d6a5f289faa02d8b70
Publikováno v:
Journal of Agricultural and Food Chemistry. 69:13020-13033
Phytoestrogens are strongly recommended for treating osteoporosis. Our previous study showed that naringin, a citrus flavonoid, can enhance the bone mass in ovariectomized rats. In this study, we further elucidated the mechanisms of naringin-induced
Publikováno v:
Molecules
Molecules, Vol 25, Iss 2876, p 2876 (2020)
Volume 25
Issue 12
Molecules, Vol 25, Iss 2876, p 2876 (2020)
Volume 25
Issue 12
An estrogen deficiency is the main cause of osteoporosis in postmenopausal women. In bone remodeling, estrogen receptors (ERs) can mediate estrogen-transducing signals. Methylpiperidinopyrazole (MPP) is a highly specific antagonist of ER-alpha (ER&al
Publikováno v:
Life sciences. 258
Aims The estrogen-ERα axis participates in osteoblast maturation. This study was designed to further evaluated the roles of the estrogen-ERα axis in bone healing and the possible mechanisms. Main methods Female ICR mice were created a metaphyseal b
Akademický článek
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Autor:
Lei Zhou, Li Wang, Kejiang Cao, Xiao-wen Zhang, Jun Huang, Jim-hong Wang, Xin Wu, Xin-Zheng Lu
Publikováno v:
Acta Pharmacologica Sinica. 29:1209-1214
To investigate the possible mechanisms of ginsenoside Rg1 promoting bone marrow stromal cell (BMSC) proliferation. BMSC were isolated from bone marrow of Sprague-Dawley rats and maintained in vitro. After stimulation with 1 μmol/L ginsenoside Rg1 fo
Objective: Neointima formation is the underlying mechanism of (in-stent) restenosis. 17β-Estradiol (E2) is known to inhibit injury-induced neointima formation and post-angioplasty restenosis. Estrogen receptor alpha (ERα) has been demonstrated to m
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=dris___00893::410802f4de9a08ea48b39c8d6bee3c88
http://resolver.tudelft.nl/uuid:a8c55a26-591a-46cf-8aee-29d878d89610
http://resolver.tudelft.nl/uuid:a8c55a26-591a-46cf-8aee-29d878d89610
Autor:
Ko Willems van Dijk, Paul H.A. Quax, J. Wouter Jukema, Margreet R. de Vries, Nuno Pires, Louis M. Havekes, Rune R. Frants, Yvonne D. Krom
Publikováno v:
Cardiovascular research. 73(1)
Objective: Neointima formation is the underlying mechanism of (in-stent) restenosis. 17β-Estradiol (E2) is known to inhibit injury-induced neointima formation and post-angioplasty restenosis. Estrogen receptor alpha (ERα) has been demonstrated to m