Zobrazeno 1 - 3
of 3
pro vyhledávání: '"metabolism [Gap Junctions]"'
Autor:
Steffen Kautzmann, Anne Boehlen, Christian Henneberger, Christian Steinhäuser, Björn Breithausen
Publikováno v:
Glia 68(5), 918-931 (2019). doi:10.1002/glia.23751
Astrocytes form large networks, in which individual cells are connected via gap junctions. It is thought that this astroglial gap junction coupling contributes to the buffering of extracellular K+ increases. However, it is largely unknown how the con
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0af3b715b32b7680e9be4af85e37d222
https://pub.dzne.de/record/141697
https://pub.dzne.de/record/141697
Autor:
Jennings, Alistair, Tyurikova, Olga, Bard, Lucie, Zheng, Kaiyu, Semyanov, Alexey, Henneberger, Christian, Rusakov, Dmitri A
Publikováno v:
Glia 65(3), 447-459 (2016). doi:10.1002/glia.23103
Whilst astrocytes in culture invariably respond to dopamine with cytosolic Ca2+ rises, the dopamine sensitivity of astroglia in situ and its physiological roles remain unknown. To minimize effects of experimental manipulations on astroglial physiolog
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od_____10678::26bb1f2a1ae32c19e8dae3f919388bef
https://pub.dzne.de/record/139131
https://pub.dzne.de/record/139131
Publikováno v:
Human Molecular Genetics, vol. 12, no. 24, pp. 3287-94
Connexins are homologous four-transmembrane-domain proteins and major components of gap junctions. We recently identified mutations in either GJB3 or GJB4 genes, encoding respectively connexin 31 (Cx31) or 30.3 (Cx30.3), as causally involved in eryth
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::40fa72efbfbfea876a80dd19fba995bb
https://pubmed.ncbi.nlm.nih.gov/14583444
https://pubmed.ncbi.nlm.nih.gov/14583444
