Zobrazeno 1 - 7
of 7
pro vyhledávání: '"metabolism [GABAergic Neurons]"'
Autor:
Stefan Remy, Christina Müller
Publikováno v:
Cell & tissue research 373(3), 565-575 (2017). doi:10.1007/s00441-017-2745-2
Cell and Tissue Research
Cell and Tissue Research
The septo-hippocampal pathway adjusts CA1 network excitability to different behavioral states and is crucially involved in theta rhythmogenesis. In the medial septum, cholinergic, glutamatergic and GABAergic neurons form a highly interconnected local
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::426eddeab25a0c468af1adc1205ad6c8
https://doi.org/10.1007/s00441-017-2745-2
https://doi.org/10.1007/s00441-017-2745-2
Publikováno v:
Brain research 1707, 216-226 (2019). doi:10.1016/j.brainres.2018.11.042
Alzheimer's disease (AD) is characterized by the classical hallmarks of Aβ-deposition and tau-pathology that are thought to ultimately lead to synapse and neuron loss. Although long known, neuroinflammation has recently attracted a substantial amoun
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6a255bc2a5307e9361577208012c6d37
https://pubmed.ncbi.nlm.nih.gov/30503351
https://pubmed.ncbi.nlm.nih.gov/30503351
Autor:
Nada Zecevic, Stewart A. Anderson, Asif M. Maroof, Albert E. Ayoub, Fani Memi, Igor Jakovcevski, Xiaojing Yu, Nevena V. Radonjić, Pasko Rakic
Publikováno v:
Cell Reports, Vol 9, Iss 6, Pp 2139-2151 (2014)
Cell reports
Cell reports 9(6), 2139-2151 (2014). doi:10.1016/j.celrep.2014.11.026
Cell reports
Cell reports 9(6), 2139-2151 (2014). doi:10.1016/j.celrep.2014.11.026
Summary Evolutionary elaboration of tissues starts with changes in the genome and location of the stem cells. For example, GABAergic interneurons of the mammalian neocortex are generated in the ventral telencephalon and migrate tangentially to the ne
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::99e8975a7593cf8c76468a32323d17c8
https://doi.org/10.1016/j.celrep.2014.11.026
https://doi.org/10.1016/j.celrep.2014.11.026
Autor:
Lucia De Franceschi, Norman Kalmbach, Hannes Glass, Verena Lübben, Nancy Stanslowsky, Jared Sterneckert, Francesca Lupo, Anna Venneri, Florian Wegner, Niko Hensel, Arun Pal, Peter Reinhardt, Maximilian Naujock, Alexander Storch, Andreas Hermann, Peter Claus
Publikováno v:
The journal of neuroscience 36(47), 12027-12043 (2016). doi:10.1523/JNEUROSCI.0456-16.2016
Chorea-acanthocytosis (ChAc) is a fatal neurological disorder characterized by red blood cell acanthocytes and striatal neurodegeneration. Recently, severe cell membrane disturbances based on depolymerized cortical actin and an elevated Lyn kinase ac
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d2f8577f2b85958466c1ea72bfe83f55
http://hdl.handle.net/11562/962218
http://hdl.handle.net/11562/962218
Autor:
Oliver Kobler, Matthias Heidenreich, Alexey Ponomarenko, Thomas J. Jentsch, Pawel Fidzinski, Dietmar Schmitz, Sebastian Schuetze, Tatiana Korotkova, Werner Zuschratter, Nikolaus Maier
Publikováno v:
Nature Communications 6(1), 6254 (2015). doi:10.1038/ncomms7254
KCNQ2 (Kv7.2) and KCNQ3 (Kv7.3) K(+) channels dampen neuronal excitability and their functional impairment may lead to epilepsy. Less is known about KCNQ5 (Kv7.5), which also displays wide expression in the brain. Here we show an unexpected role of K
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c6290d8981a828ce765e5b932dcaaaca
https://pub.dzne.de/record/137796
https://pub.dzne.de/record/137796
Autor:
Jochen Herms, Stylianos Michalakis, Saak V. Ovsepian, Mario M. Dorostkar, Martin Biel, Severin Filser, Aayush Gupta, Johanna Neuner
Publikováno v:
Nature Communications
Nature Communications 5(1), 3915 (2014). doi:10.1038/ncomms4915
Nature Communications 5(1), 3915 (2014). doi:10.1038/ncomms4915
Although the role of noxious α-synuclein (α-SYN) in the degeneration of midbrain dopaminergic neurons and associated motor deficits of Parkinson’s disease is recognized, its impact on non-motor brain circuits and related symptoms remains elusive.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8532396fde93a66dccedd0d972b5eabf
http://europepmc.org/articles/PMC4050256
http://europepmc.org/articles/PMC4050256
Autor:
Holger Lerche, Martin Pofahl, Stephan Theiss, Camille Liautard, Yuanyuan Liu, Johannes Slotta, Ulrike B. S. Hedrich, Andrew Escayg, Jennifer Lavigne, Heinz Beck, Massimo Mantegazza, Marcel Dihné, Daniel Kirschenbaum
Publikováno v:
The journal of neuroscience 34(45), 14874-14889 (2014). doi:10.1523/JNEUROSCI.0721-14.2014
Mutations inSCN1Aand other ion channel genes can cause different epileptic phenotypes, but the precise mechanisms underlying the development of hyperexcitable networks are largely unknown. Here, we present a multisystem analysis of anSCN1Amouse model
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fcc8901192a48f0f5e7d5e96533da9d3