Activation of the AMP-related kinase (AMPK) induces renal vasodilatation and downregulates Nox-derived reactive oxygen species (ROS) generation

Autor: Claudia Rodríguez, Cristina Contreras, Javier Sáenz-Medina, Mercedes Muñoz, César Corbacho, Joaquín Carballido, Albino García-Sacristán, Medardo Hernandez, Miguel López, Luis Rivera, Dolores Prieto

Publikováno v: Redox Biology, Vol 34, Iss , Pp 101575- (2020)

AMP-activated protein kinase (AMPK) is a cellular energy sensor activated during energy stress to stimulate ATP production pathways and restore homeostasis. AMPK is widely expressed in the kidney and involved in mitochondrial protection and biogenesi

Externí odkaz: https://doaj.org/article/45834cd73fe84a8b9bd4b67b363fae89

Activation of the AMP-related kinase (AMPK) induces renal vasodilatation and downregulates Nox-derived reactive oxygen species (ROS) generation

Autor: Albino García-Sacristán, Miguel López, Luis Rivera, César Corbacho, Javier Sáenz-Medina, Claudia Rodríguez, Mercedes Muñoz, Joaquín Carballido, Dolores Prieto, Cristina Contreras, Medardo Hernández

Publikováno v: Redox Biology, Vol 34, Iss, Pp 101575-(2020)
Redox Biology

AMP-activated protein kinase (AMPK) is a cellular energy sensor activated during energy stress to stimulate ATP production pathways and restore homeostasis. AMPK is widely expressed in the kidney and involved in mitochondrial protection and biogenesi

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5e7ea2e2b0a93197fbe6edf320446cb7
http://www.sciencedirect.com/science/article/pii/S2213231720305541

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The Amyloid Precursor Protein C-Terminal Domain Alters CA1 Neuron Firing, Modifying Hippocampus Oscillations and Impairing Spatial Memory Encoding

Autor: Sebastian P. Fernandez, C. Gandin, Xavier Mouska, Ana Rita Salgueiro-Pereira, Daniela Bianchi, Rui Gomes, Mariana Temido-Ferreira, Luísa V. Lopes, Elisabeth F. Raymond, Michele Migliore, Jacques Barik, Hélène Marie, Ingrid Bethus, Joana Rajão-Saraiva, Paula A. Pousinha, Romain Goutagny

Publikováno v: Cell Reports
Cell reports 29 (2019): 317–+. doi:10.1016/j.celrep.2019.08.103
info:cnr-pdr/source/autori:Pousinha, Paula A.; Mouska, Xavier; Bianchi, Daniela; Temido-Ferreira, Mariana; Rajao-Saraiva, Joana; Gomes, Rui; Fernandez, Sebastian P.; Salgueiro-Pereira, Ana Rita; Gandin, Carine; Raymond, Elisabeth F.; Barik, Jacques; Goutagny, Romain; Bethus, Ingrid; Lopes, Luisa V.; Migliore, Michele; Marie, Helene/titolo:The Amyloid Precursor Protein C-Terminal Domain Alters CA1 Neuron Firing, Modifying Hippocampus Oscillations and Impairing Spatial Memory Encoding/doi:10.1016%2Fj.celrep.2019.08.103/rivista:Cell reports/anno:2019/pagina_da:317/pagina_a:+/intervallo_pagine:317–+/volume:29
Cell Reports, Elsevier Inc, 2019, 29 (2), pp.317-331.e5. ⟨10.1016/j.celrep.2019.08.103⟩
Cell Reports, Vol 29, Iss 2, Pp 317-331.e5 (2019)

Summary: There is a growing consensus that Alzheimer's disease (AD) involves failure of the homeostatic machinery, which underlies the firing stability of neural circuits. What are the culprits leading to neuron firing instability? The amyloid precur

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ca3775443dadc20459dfd41c39d8afa2
https://doi.org/10.1016/j.celrep.2019.08.103

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