Zobrazeno 1 - 6
of 6
pro vyhledávání: '"fas Receptor/metabolism"'
Autor:
Nicolas Degauque, Claire Soulard, Frédérique Scamps, David Laplaud, Attila Gergely Végh, Céline Salsac, Margot Libralato, Cédric Raoul, Emmanuelle Coque, Stéphanie Ventéo, Csilla Gergely, José Boucraut, Gabriel Espinosa-Carrasco, Roxane Crabé, Béla Varga, Alexandre Brodovitch, Thierry Vincent, Anaïs Virenque, Javier Hernandez, Julie K. Fierle, Marion Cadoux
Publikováno v:
Proceedings of the National Academy of Sciences of the United States of America
Proceedings of the National Academy of Sciences of the United States of America, National Academy of Sciences, 2019, 116 (6), pp.2312-2317. ⟨10.1073/pnas.1815961116⟩
Proceedings of the National Academy of Sciences of the United States of America, 2019, 116 (6), pp.2312-2317. ⟨10.1073/pnas.1815961116⟩
Proceedings of the National Academy of Sciences of the United States of America, vol. 116, no. 6, pp. 2312-2317
Proceedings of the National Academy of Sciences of the United States of America, National Academy of Sciences, 2019, 116 (6), pp.2312-2317. ⟨10.1073/pnas.1815961116⟩
Proceedings of the National Academy of Sciences of the United States of America, 2019, 116 (6), pp.2312-2317. ⟨10.1073/pnas.1815961116⟩
Proceedings of the National Academy of Sciences of the United States of America, vol. 116, no. 6, pp. 2312-2317
Adaptive immune response is part of the dynamic changes that accompany motoneuron loss in amyotrophic lateral sclerosis (ALS). CD4 + T cells that regulate a protective immunity during the neurodegenerative process have received the most attention. CD
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c9b74d69287c1970bf57c427665e4698
https://www.hal.inserm.fr/inserm-03171288
https://www.hal.inserm.fr/inserm-03171288
Autor:
Gagnoux-Palacios, Laurent, Awina, Hala, Audebert, Stéphane, Rossin, Aurélie, Mondin, Magali, Borgese, Franck, Planas-Botey, Carlota, Mettouchi, Amel, Borg, Jean-Paul, Hueber, Anne-Odile
Publikováno v:
Journal of Cell Biology
Journal of Cell Biology, 2018, 217 (11), pp.3839-3852. ⟨10.1083/jcb.201805071⟩
The Journal of Cell Biology
Journal of Cell Biology, Rockefeller University Press, 2018, 217 (11), pp.3839-3852. ⟨10.1083/jcb.201805071⟩
Journal of Cell Biology, 2018, 217 (11), pp.3839-3852. ⟨10.1083/jcb.201805071⟩
The Journal of Cell Biology
Journal of Cell Biology, Rockefeller University Press, 2018, 217 (11), pp.3839-3852. ⟨10.1083/jcb.201805071⟩
Control of epithelial cell death is crucial to maintaining tissue integrity. Gagnoux-Palacios et al. show that cell polarity and adherens junction formation prevent proapoptotic signals emanating from the Fas death receptor. Therefore, Fas-dependent
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::5b791786ff71a152338fb2a61cdb54f7
https://hal.science/hal-02374965/document
https://hal.science/hal-02374965/document
Autor:
Andy I. M. Hoepelman, F. J. P. Höppener, K. R. Hirsch, Joop E. Arends, Nening M. Nanlohy, J. G. Park, D. van Baarle
Publikováno v:
Apoptosis, 16(9), 959-66
Apoptosis
Apoptosis
Chronic hepatitis C virus (HCV) infection is characterized by increased rates of apoptotic hepatocytes and activated caspases have been shown in HCV-infected patients. GS-9450, a novel caspase-inhibitor has demonstrated hepatoprotective activity in f
Autor:
Laurent Spahr, Laura Rubbia-Brandt, Rafael Quadri, Karim Abid, Patrizia Gindre, Sophia Taylor, Francesco Negro
Publikováno v:
The Journal of pathology, vol. 197, no. 5, pp. 617-623
In vitro data have shown that the hepatitis C virus (HCV) core protein binds to protein members of the tumour necrosis factor receptor (TNFR) superfamily. Since this interaction could be relevant to HCV persistence and oncogenesis, this study assesse
Autor:
M Antonietta, Medici, M Teresa, Sciortino, Donata, Perri, Carla, Amici, Elisa, Avitabile, Marco, Ciotti, Emanuela, Balestrieri, Enrico, De Smaele, Guido, Franzoso, Antonio, Mastino
Signals involved in protection against apoptosis by herpes simplex virus 1 (HSV-1) were investigated. Using U937 monocytoid cells as an experimental model, we have demonstrated that HSV-1 rendered these cells resistant to Fas-induced apoptosis prompt
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::e83c1281ae4d90822b432e16709150d5
http://hdl.handle.net/2108/65873
http://hdl.handle.net/2108/65873
Autor:
Ying Liu, Ramona Pop, Merav Socolovsky, Ermelinda Porpiglia, Michael P. Murrell, Andre Levchenko
Publikováno v:
PLoS Biology, Vol 5, Iss 10, p e252 (2007)
PLoS Biology
Socolovsky, M, Murrell, M, Liu, Y, Pop, R, Porpiglia, E & Levchenko, A 2007, ' Negative autoregulation by FAS mediates robust fetal erythropoiesis ', PLoS Biology, vol. 5, no. 10, pp. e252 . https://doi.org/10.1371/journal.pbio.0050252
PLoS Biology
Socolovsky, M, Murrell, M, Liu, Y, Pop, R, Porpiglia, E & Levchenko, A 2007, ' Negative autoregulation by FAS mediates robust fetal erythropoiesis ', PLoS Biology, vol. 5, no. 10, pp. e252 . https://doi.org/10.1371/journal.pbio.0050252
Tissue development is regulated by signaling networks that control developmental rate and determine ultimate tissue mass. Here we present a novel computational algorithm used to identify regulatory feedback and feedforward interactions between progen