Zobrazeno 1 - 4
of 4
pro vyhledávání: '"drug effects [Oligodendroglia]"'
Autor:
Stephan A. Müller, Maria Inês Cunha, Stefan F. Lichtenthaler, Anne Winkler, Bettina Schmid, Franziska van der Meer, Tjakko J. van Ham, Ioannis Alexopoulos, Mikael Simons, Minhui Su, Minou Djannatian, Christine Stadelmann, Martina Schifferer, Ludovico Cantuti-Castelvetri
Publikováno v:
Journal of Experimental Medicine, 217(5):e20191390. Rockefeller University Press
The Journal of Experimental Medicine
Journal of experimental medicine 217(5), e20191390 (2020). doi:10.1084/jem.20191390
Journal of Experimental Medicine
The Journal of Experimental Medicine
Journal of experimental medicine 217(5), e20191390 (2020). doi:10.1084/jem.20191390
Journal of Experimental Medicine
We find that pro-inflammatory activation is required for remyelination after myelin injury. We impaired inflammatory signaling and found that microglia in lesioned animals were defective in phagosome maturation, debris clearance, and also in triggeri
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f8afc169c9da206914a69fcfd5c9cbd3
https://pure.eur.nl/en/publications/92b7bc9e-deb4-4a4e-9a67-9b4171d74756
https://pure.eur.nl/en/publications/92b7bc9e-deb4-4a4e-9a67-9b4171d74756
Autor:
Mikael Simons, Ludovico Cantuti-Castelvetri, Florian Raabe, Moritz J. Rossner, Andrea Schmitt, Lenka Slapakova, Peter Falkai
Publikováno v:
Cells 8(12), 1496-(2019). doi:10.3390/cells8121496
Cells
Cells
Imaging and postmortem studies have revealed disturbed oligodendroglia-related processes in patients with schizophrenia and provided much evidence for disturbed myelination, irregular gene expression, and altered numbers of oligodendrocytes in the br
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2144e003878fd623323c29e9fe83516f
Autor:
Frühbeis, C., Fröhlich, D., Kuo, W., Amphornrat, J., Thilemann, S., Saab, A., Kirchhoff, F., Möbius, W., Goebbels, S., Nave, K., Schneider, A., Simons, M., Klugmann, M., Trotter, J., Krämer-Albers, E.
Publikováno v:
PLoS Biology, Vol 11, Iss 7, p e1001604 (2013)
PLoS Biology
PLoS biology 11(7), e1001604 (2013). doi:10.1371/journal.pbio.1001604
PLoS Biology
PLoS biology 11(7), e1001604 (2013). doi:10.1371/journal.pbio.1001604
Neuronal activity provokes myelinating oligodendrocytes to release exosomes by stimulation of ionotropic glutamate receptors, and that once released, these vesicles are internalized by neurons conveying neuroprotection.
Reciprocal interactions b
Reciprocal interactions b
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ac6dc5a77cd5972dbc7705feafec3b74
https://www.ncbi.nlm.nih.gov/pmc/articles/pmid/23874151/?tool=EBI
https://www.ncbi.nlm.nih.gov/pmc/articles/pmid/23874151/?tool=EBI
FAS-dependent cell death in α-synuclein transgenic oligodendrocyte models of multiple system atrophy
Autor:
Manuela Neumann, Glenda M. Halliday, Hanne D. Hansen, Wei Ping Gai, Christiane Richter-Landsberg, Christian Haass, Christine Lund Kragh, Catherine Lubetzki, Amanda M. Gysbers, Poul Henning Jensen, Bernard Zalc, Gwenaelle Fillon, Philipp J. Kahle
Publikováno v:
PLOS ONE 8(1), e55243 (2013). doi:10.1371/journal.pone.0055243
PLoS ONE, Vol 8, Iss 1, p e55243 (2013)
PLoS ONE; Vol 8
Kragh, C L, Fillon, G, Gysbers, A, Hansen, H D, Neumann, M, Richter-Landsberg, C, Haass, C, Zalc, B, Lubetzki, C, Gai, W-P, Halliday, G M, Kahle, P J & Jensen, P H 2013, ' FAS-Dependent Cell Death in α-Synuclein Transgenic Oligodendrocyte Models of Multiple System Atrophy ', P L o S One, vol. 8, no. 1, pp. e55243 . https://doi.org/10.1371/journal.pone.0055243
PLoS ONE
PLoS ONE, Public Library of Science, 2013, 8 (1), ⟨10.1371/journal.pone.0055243⟩
PLoS ONE, Vol 8, Iss 1, p e55243 (2013)
PLoS ONE; Vol 8
Kragh, C L, Fillon, G, Gysbers, A, Hansen, H D, Neumann, M, Richter-Landsberg, C, Haass, C, Zalc, B, Lubetzki, C, Gai, W-P, Halliday, G M, Kahle, P J & Jensen, P H 2013, ' FAS-Dependent Cell Death in α-Synuclein Transgenic Oligodendrocyte Models of Multiple System Atrophy ', P L o S One, vol. 8, no. 1, pp. e55243 . https://doi.org/10.1371/journal.pone.0055243
PLoS ONE
PLoS ONE, Public Library of Science, 2013, 8 (1), ⟨10.1371/journal.pone.0055243⟩
Multiple system atrophy is a parkinsonian neurodegenerative disorder. It is cytopathologically characterized by accumulation of the protein p25α in cell bodies of oligodendrocytes followed by accumulation of aggregated α-synuclein in so-called glia
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a3eec5b7d30bcecf6ed2099bbeac7e31
https://pub.dzne.de/record/136805
https://pub.dzne.de/record/136805