Zobrazeno 1 - 10
of 338
pro vyhledávání: '"di Mario, Umberto"'
Autor:
Tiberti, Claudio, Verrienti, Antonella, Fiore, Benedetta, Yu, Liping, Eisenbarth, George S., Dotta, Francesco, Di Mario, Umberto
Publikováno v:
In Clinical Immunology 2005 115(3):260-267
Autor:
Iacobellis, Gianluca, Cipriani, Rosalba, Gabriele, Annarita, Di Mario, Umberto, Morano, Susanna
Publikováno v:
In Cytokine 2004 27(1):25-30
Autor:
Sorropago Giovanni, Tesorio Tullio, Arca Marcello, Romeo Stefano, Berni Andrea, Baroni Marco G, Di Mario Umberto, Galton David J
Publikováno v:
BMC Medical Genetics, Vol 4, Iss 1, p 8 (2003)
Abstract Background Current evidence demonstrates that positive family history and several alterations in lipid metabolism are all important risk factors for coronary artery disease (CAD). All lipid abnormalities themselves have genetic determinants.
Externí odkaz:
https://doaj.org/article/c218a7fe4fde425cb6a4cd2efdd56aea
Autor:
Pricci, Flavia, Leto, Gaetano, Amadio, Lorena, Iacobini, Carla, Romeo, Giulio, Cordone, Samantha, Gradini, Roberto, Barsotti, Paola, Liu, Fu-Tong, Di Mario, Umberto, Pugliese, Giuseppe *
Publikováno v:
In Kidney International September 2000 58 Supplement 77:S31-S39
Autor:
Vecci Elio, Berni Andrea, Arca Marcello, Capici Francesca, Sentinelli Federica, Romeo Stefano, Di Mario Umberto, Baroni Marco
Publikováno v:
BMC Medical Genetics, Vol 2, Iss 1, p 10 (2001)
Abstract Background Tumor Necrosis Factor-α (TNF-α) has been implicated in the pathogenesis of insulin resistance and obesity. The increased expression of TNF-α in adipose tissue has been shown to induce insulin resistance, and a polymorphism at p
Externí odkaz:
https://doaj.org/article/532b843b46524fcabecb2907a782c71d
Autor:
Morano, Susanna *, Tiberti, Claudio, Cristina, Giuseppe, Sensi, Maurizio, Cipriani, Rosalba, Guidobaldi, Leo, Torresi, Patrizia, Medici, Francesco, Anastasi, Emanuela, Di Mario, Umberto
Publikováno v:
In Human Immunology 1999 60(9):848-854
Autor:
DI MARIO, Umberto, Pugliese, Giuseppe
Publikováno v:
International Congress Series. 1253:171-182
Hyperglycaemia has been shown to play a central role in diabetic microangiopathy, together with the individual background. The pathogenetic sequence is initiated by a series of interrelated biochemical abnormalities associated with hyperglycaemia, in