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Akademický článek

Neuron-specific knockdown of solute carrier protein SLC25A46a induces locomotive defects, an abnormal neuron terminal morphology, learning disability, and shortened lifespan

Autor: Md Saheb Ali, Kojiro Suda, Ryosuke Kowada, Ibuki Ueoka, Hideki Yoshida, Masamitsu Yamaguchi

Publikováno v: IBRO Reports, Vol 8, Iss , Pp 65-75 (2020)

Various mutations in the SLC25A46 gene have been reported in mitochondrial diseases that are sometimes classified as type 2 Charcot-Marie-Tooth disease, optic atrophy, and Leigh syndrome. Although human SLC25A46 is a well-known transporter that acts

Externí odkaz: https://doaj.org/article/f30c379a724045a6bd5d5b1173e52df0

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Neuron-specific knockdown of solute carrier protein SLC25A46a induces locomotive defects, an abnormal neuron terminal morphology, learning disability, and shortened lifespan

Autor: Ryosuke Kowada, Saheb Ali, Kojiro Suda, Ibuki Ueoka, Hideki Yoshida, Masamitsu Yamaguchi

Publikováno v: IBRO Reports, Vol 8, Iss, Pp 65-75 (2020)
IBRO Reports

Highlights • Neuron-specific dSLC25A46a knockdown resulted in reduced mobility in larvae as well as adults. • Neuron-specific dSLC25A46a knockdown induced an aberrant morphology for NMJ and learning disability. • Neuron-specific dSLC25A46a knoc

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::96062f6d45c7d6731f58c10301910b28
http://www.sciencedirect.com/science/article/pii/S2451830120300054

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Akademický článek

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