Zobrazeno 1 - 10
of 35
pro vyhledávání: '"conditional transgenic mouse"'
Autor:
Georgia A. Frentzou, Mark J. Drinkhill, Neil A. Turner, Stephen G. Ball, Justin F. X. Ainscough
Publikováno v:
Disease Models & Mechanisms, Vol 8, Iss 8, Pp 783-794 (2015)
Cardiac dysfunction is commonly associated with high-blood-pressure-induced cardiomyocyte hypertrophy, in response to aberrant renin-angiotensin system (RAS) activity. Ensuing pathological remodelling promotes cardiomyocyte death and cardiac fibrobla
Externí odkaz:
https://doaj.org/article/83c9df25c2c744218297d9fdc6896a07
Autor:
Yusuke Murasawa, Ken Watanabe, Shumpei Niida, Hiroyuki Nakamura, Tomoko Hayakawa, Taiki Mori, Rina Ikai, Noboru Ogiso
Publikováno v:
Experimental Animals
IL-6 is a cytokine that is involved in various physiological and pathological conditions, and approaches using gain-of-function transgenic animals have contributed in elucidating IL-6 function. However, studies of the multiple functions of IL-6 in vi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6c9526d998cd3ca3752142805ae48a03
https://doi.org/10.1538/expanim.16-0043
https://doi.org/10.1538/expanim.16-0043
Publikováno v:
Disease Models & Mechanisms, Vol 8, Iss 8, Pp 783-794 (2015)
Cardiac dysfunction is commonly associated with high-blood-pressure-induced cardiomyocyte hypertrophy, in response to aberrant renin-angiotensin system (RAS) activity. Ensuing pathological remodelling promotes cardiomyocyte death and cardiac fibrobla
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=dedup_wf_001::d2d1cc96fdcafd8b40e8bc44df0aeb45
http://dmm.biologists.org/content/8/8/783
http://dmm.biologists.org/content/8/8/783
Akademický článek
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Autor:
Georgia A, Frentzou, Mark J, Drinkhill, Neil A, Turner, Stephen G, Ball, Justin F X, Ainscough
Publikováno v:
Disease Models & Mechanisms
Cardiac dysfunction is commonly associated with high-blood-pressure-induced cardiomyocyte hypertrophy, in response to aberrant renin-angiotensin system (RAS) activity. Ensuing pathological remodelling promotes cardiomyocyte death and cardiac fibrobla
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::4dc7085fc64de702dabf49c0dcae0c44
https://pubmed.ncbi.nlm.nih.gov/26092119
https://pubmed.ncbi.nlm.nih.gov/26092119
Autor:
Mara Pietrobon, Giuseppe Cifelli, Philip J. Gage, Michael D. Schneider, Marina Campione, Antonella Notte, Alessandra Tessari, Giuseppe Lembo
Publikováno v:
Circulation Research
The Pitx2 gene regulates left-right (L/R) asymmetrical cardiac morphogenesis. Constitutive Pitx2 knock out (ko) mice die before birth and display, among other defects, right atrial isomerism, atrial and ventricular septal defects, and double outlet r
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d98e5641af8e91fadd777f98b8af1096
http://hdl.handle.net/11573/369026
http://hdl.handle.net/11573/369026
Autor:
Ester Martín-Aparicio, Miguel Díaz-Hernández, Isidro Ferrer, Félix Hernández, María A. Morán, José J. Lucas, José G. Castaño, Jesús Avila, Pilar Gómez-Ramos
Publikováno v:
ResearcherID
Dipòsit Digital de la UB
Universidad de Barcelona
Europe PubMed Central
Recercat. Dipósit de la Recerca de Catalunya
instname
Scopus-Elsevier
Digital.CSIC. Repositorio Institucional del CSIC
Dipòsit Digital de la UB
Universidad de Barcelona
Europe PubMed Central
Recercat. Dipósit de la Recerca de Catalunya
instname
Scopus-Elsevier
Digital.CSIC. Repositorio Institucional del CSIC
Huntington's disease (HD) inclusions are stained with anti-ubiquitin and anti-proteasome antibodies. This, together with proteasome activity studies on transfected cells, suggest that an impairment of the ubiquitin-proteasome system (UPS) may be key
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0a3592fd7a1adc7b7db2048ee1a813c9
https://pubmed.ncbi.nlm.nih.gov/14684867
https://pubmed.ncbi.nlm.nih.gov/14684867
Autor:
Maddalena T. Tilli, Priscilla A. Furth
Publikováno v:
Breast Cancer Research
Diversity in the pathophysiology of breast cancer frustrates therapeutic progress. We need to understand how mechanisms activated by specific combinations of oncogenes, tumor suppressors, and hormonal signaling pathways govern response to therapy and
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::939c735dd795e89e01ada91589e4e1b0
https://doi.org/10.1186/bcr614
https://doi.org/10.1186/bcr614
Akademický článek
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Akademický článek
Tento výsledek nelze pro nepřihlášené uživatele zobrazit.
K zobrazení výsledku je třeba se přihlásit.
K zobrazení výsledku je třeba se přihlásit.