Zobrazeno 1 - 10
of 886
pro vyhledávání: '"cholesterol crystals"'
Autor:
Kensuke Nishimiya, Gargi Sharma, Kanwarpal Singh, Osman O. Ahsen, Joseph A. Gardecki, Guillermo J. Tearney
Publikováno v:
Frontiers in Cardiovascular Medicine, Vol 11 (2024)
IntroductionBirefringent crystals such as monosodium-urate (MSU) and cholesterol crystals (CC) likely contribute to the progression of coronary artery disease (CAD) due to their potential to exacerbate inflammation through inflammatory cytokine activ
Externí odkaz:
https://doaj.org/article/38311d41d38a4d9bbd02cddcb037dc41
Autor:
Yi Shi, Meige Zheng, Yang Luo, Jianjian Li, Fangru Ouyang, Yuanzhe Zhao, Jingwen Wang, Zhida Ma, Congpeng Meng, Yihui Bi, Li Cheng, Juehua Jing
Publikováno v:
Frontiers in Neuroscience, Vol 18 (2024)
BackgroundAfter spinal cord injury (SCI), lipid metabolism dysregulation at the lesion site exacerbates secondary damage. The transcription factor pu.1 has been implicated as a negative regulator of multiple lipid metabolism-related genes and pathway
Externí odkaz:
https://doaj.org/article/282d7e08bc434e658266afa936460186
Publikováno v:
Frontiers in Cardiovascular Medicine, Vol 11 (2024)
BackgroundInflammation is pivotal to the progression of atherosclerosis. Cholesterol crystals (CCs) that grow and enlarge within the plaque core can cause plaque rupture and trigger inflammation as they deposit into the atherosclerotic bed. Thus, age
Externí odkaz:
https://doaj.org/article/13cb816d9028463e903fd2cd08a697cc
Autor:
Baravan Al‐Kassou, Lara Al‐Kassou, Thorsten Mahn, Dieter Lütjohann, Jasmin Shamekhi, Nicola Willemsen, Sven Thomas Niepmann, Stephan Baldus, Malte Kelm, Georg Nickenig, Eicke Latz, Sebastian Zimmer
Publikováno v:
Journal of the American Heart Association: Cardiovascular and Cerebrovascular Disease, Vol 13, Iss 3 (2024)
Background Aortic stenosis has pathophysiological similarities with atherosclerosis, including the deposition of cholesterol‐containing lipoproteins. The resulting cholesterol crystals activate the NLRP3 (NOD‐like receptor protein 3) inflammasome
Externí odkaz:
https://doaj.org/article/8256c019cdd446ceb39c342897b4d23c
Autor:
George S. Abela, Venkat R. Katkoori, Dorothy R. Pathak, Harvey L. Bumpers, Monika Leja, Zain ul Abideen, Manel Boumegouas, Daniel Perry, Anas Al-Janadi, James E. Richard, Carlo Barnaba, Ilce G. Medina Meza
Publikováno v:
American Heart Journal Plus, Vol 35, Iss , Pp 100317- (2023)
Background and aims: Cancer and atherosclerosis share common risk factors and inflammatory pathways that promote their proliferation via vascular endothelial growth factor (VEGF). Because CCs cause mechanical injury and inflammation in atherosclerosi
Externí odkaz:
https://doaj.org/article/c1ab1b81aae94a41a9d5fe8b46532081
Akademický článek
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Autor:
Chao Xue, Qizhi Chen, Ling Bian, Zhaofang Yin, Zuojun Xu, Huili Zhang, Qingyong Zhang, Junfeng Zhang, Changqian Wang, Run Du, Li Fan
Publikováno v:
Frontiers in Cardiovascular Medicine, Vol 9 (2022)
BackgroundCholesterol crystals (CCs) in lesions are the hallmark of advanced atherosclerotic plaque. Previous studies have demonstrated that CCs could activate NLRP3 inflammasome, which played an important role in atherosclerotic lesion progression.
Externí odkaz:
https://doaj.org/article/11a7c89f9a4949d2b6f99f8cece8b5eb
Akademický článek
Tento výsledek nelze pro nepřihlášené uživatele zobrazit.
K zobrazení výsledku je třeba se přihlásit.
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Autor:
Sei Komatsu, MD, PhD, Chikao Yutani, MD, PhD, Mitsuhiko Takewa, MD, PhD, Satoru Takahashi, MD, Kazuhisa Kodama, MD, PhD
Publikováno v:
JACC: Case Reports, Vol 2, Iss 4, Pp 615-618 (2020)
Spontaneous cholesterol embolization is thought to be due to aortic plaque-derived shower emboli; however, flowing cholesterol crystals have not been detected. Free multilayer cholesterol crystals obtained from the femoral artery were observed using
Externí odkaz:
https://doaj.org/article/4e21f0c48cf24ecf93f60fdf5cf4830f
Publikováno v:
American Heart Journal Plus, Vol 13, Iss , Pp 100083- (2022)
Background and aims: Cholesterol crystals (CCs) have been found to be critical in the evolution and progression of atherosclerotic plaque leading up to rupture. This includes triggering inflammation and mechanically traumatizing the plaque and surrou
Externí odkaz:
https://doaj.org/article/44563547ea7a4da4b3e1ff77f65024c1