Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Zuzana Kubalova"'
Autor:
Pompeo Volpe, Srikanth Vedamoorthyrao, Serge Viatchenko-Karpinski, Alessandra Nori, Giorgia Valle, Radmila Terentyeva, Zuzana Kubalova, Dmitry Terentyev, Donald M. Bers, Sandor Gyorke, Simon C. Williams
Publikováno v:
Biophysical Journal. 95(4):2037-2048
Cardiac calsequestrin (CASQ2) is an intrasarcoplasmic reticulum (SR) low-affinity Ca-binding protein, with mutations that are associated with catecholamine-induced polymorphic ventricular tachycardia (CPVT). To better understand how CASQ2 mutants cau
Autor:
Robert L. Hamlin, Andriy E. Belevych, Dmitry Terentyev, Zuzana Kubalova, Sandor Gyorke, Cynthia A. Carnes
Publikováno v:
Biophysical Journal. 93:4083-4092
In this study, we investigated the role of elevated sarcoplasmic reticulum (SR) Ca(2+) leak through ryanodine receptors (RyR2s) in heart failure (HF)-related abnormalities of intracellular Ca(2+) handling, using a canine model of chronic HF. The cyto
Autor:
Radmila Terentyeva, Dmitry Terentyev, Inna Györke, Simon C. Williams, Serge Viatchenko-Karpinski, Zuzana Kubalova, Sandor Györke
Publikováno v:
The Journal of Physiology. 561:515-524
Waves of Ca2+-induced Ca2+ release occur in various cell types and are involved in the pathology of certain forms of cardiac arrhythmia. These arrhythmias include catecholaminergic polymorphic ventricular tachycardia (CPVT), certain cases of which ar
Autor:
Dmitry Terentyev, Srikanth Vedamoorthyrao, Inna Györke, Serge Viatchenko-Karpinski, Giorgia Valle, Massimo Santoro, Nico A. Blom, Zuzana Kubalova, Carlo Napolitano, Radmila Terentyeva, Pompeo Volpe, Simon C. Williams, Alessandra Nori, Silvia G. Priori, Sandor Gyorke
Publikováno v:
Circulation research, 98(9), 1151-1158. Lippincott Williams and Wilkins
Catecholaminergic polymorphic ventricular tachycardia (CPVT) is a familial arrhythmogenic disorder associated with mutations in the cardiac ryanodine receptor ( RyR2 ) and cardiac calsequestrin ( CASQ2 ) genes. Previous in vitro studies suggested tha
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f5d4c8b76708d89f2d2e28cb282083d0
https://pure.amc.nl/en/publications/abnormal-interactions-of-calsequestrin-with-the-ryanodine-receptor-calcium-release-channel-complex-linked-to-exerciseinduced-sudden-cardiac-death(c83a97f5-5aa1-4afa-9887-7579053b92e4).html
https://pure.amc.nl/en/publications/abnormal-interactions-of-calsequestrin-with-the-ryanodine-receptor-calcium-release-channel-complex-linked-to-exerciseinduced-sudden-cardiac-death(c83a97f5-5aa1-4afa-9887-7579053b92e4).html
Autor:
Sandor Gyorke, Robert L. Hamlin, Cynthia A. Carnes, Arun Sridhar, Yoshinori Nishijima, Serge Viatchenko-Karpinski, David S. Feldman, Inna Györke, Radmila Terentyeva, Zuzana Kubalova, Daise da Cunha, Dmitry Terentyev
Publikováno v:
Proceedings of the National Academy of Sciences of the United States of America. 102(39)
Diminished Ca release from the sarcoplasmic reticulum (SR) is an important contributor to the impaired contractility of the failing heart. Despite extensive effort, the underlying causes of abnormal SR Ca release in heart failure (HF) remain unknown.
Autor:
Zuzana, Kubalova, Inna, Györke, Radmila, Terentyeva, Serge, Viatchenko-Karpinski, Dmitry, Terentyev, Simon C, Williams, Sandor, Györke
Publikováno v:
The Journal of physiology. 561(Pt 2)
Waves of Ca2+-induced Ca2+ release occur in various cell types and are involved in the pathology of certain forms of cardiac arrhythmia. These arrhythmias include catecholaminergic polymorphic ventricular tachycardia (CPVT), certain cases of which ar
Publikováno v:
American journal of physiology. Cell physiology. 286(2)
In mammalian cardiac myocytes, calcium released into the dyadic space rapidly inactivates calcium current ( ICa). We used this Ca2+ release-dependent inactivation (RDI) of ICa as a local probe of sarcoplasmic reticulum Ca2+ release activation. In who
Publikováno v:
Neurophysiology. 32:214-214