Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Zsolt, Combi"'
Autor:
Zsolt Combi, László Potor, Péter Nagy, Katalin Éva Sikura, Tamás Ditrói, Eszter Petra Jurányi, Klaudia Galambos, Tamás Szerafin, Péter Gergely, Matthew Whiteman, Roberta Torregrossa, Yuchao Ding, Lívia Beke, Zoltán Hendrik, Gábor Méhes, György Balla, József Balla
Publikováno v:
Redox Biology, Vol 60, Iss , Pp 102629- (2023)
Hydrogen sulfide (H2S) was previously revealed to inhibit osteoblastic differentiation of valvular interstitial cells (VICs), a pathological feature in calcific aortic valve disease (CAVD). This study aimed to explore the metabolic control of H2S lev
Externí odkaz:
https://doaj.org/article/a3796fa1603843a4bad5792c6dc90283
Autor:
Katalin Éva Sikura, Zsolt Combi, László Potor, Tamás Szerafin, Zoltán Hendrik, Gábor Méhes, Péter Gergely, Matthew Whiteman, Lívia Beke, Ibolya Fürtös, György Balla, József Balla
Publikováno v:
Journal of Advanced Research, Vol 27, Iss , Pp 165-176 (2021)
Introduction: Hydrogen sulfide (H2S) was revealed to inhibit aortic valve calcification and inflammation was implicated in the pathogenesis of calcific aortic valve disease (CAVD). Objectives: We investigate whether H2S inhibits mineralization via ab
Externí odkaz:
https://doaj.org/article/bebf49e1eaec4503b17f19f754ce6c93
Autor:
László Potor, József Balla, Katalin Éva Sikura, Ibolya Fürtös, Zoltán Hendrik, Zsolt Combi, Péter Gergely, György Balla, Matthew Whiteman, Gábor Méhes, Tamás Szerafin, Lívia Beke
Publikováno v:
Journal of Advanced Research
Journal of Advanced Research, Vol 27, Iss, Pp 165-176 (2021)
Journal of Advanced Research, Vol 27, Iss, Pp 165-176 (2021)
Graphical abstract
Highlights • H2S derived from CSE/CBS inhibits inflammation and calcification of valvular interstitial cells. • H2S releasing molecules inhibits inflammation and calcification of valvular interstitial cells. • H2S inhibi
Highlights • H2S derived from CSE/CBS inhibits inflammation and calcification of valvular interstitial cells. • H2S releasing molecules inhibits inflammation and calcification of valvular interstitial cells. • H2S inhibi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2577134f86e4d61d605b2cf8a2cb940a
http://europepmc.org/articles/PMC7728582
http://europepmc.org/articles/PMC7728582
Oxidation of Hemoglobin Drives a Proatherogenic Polarization of Macrophages in Human Atherosclerosis
Autor:
László, Potor, Zoltán, Hendrik, Andreas, Patsalos, Éva, Katona, Gábor, Méhes, Szilárd, Póliska, Éva, Csősz, Gergő, Kalló, István, Komáromi, Zsolt, Combi, Niké, Posta, Katalin Éva, Sikura, Dávid, Pethő, Melinda, Oros, György, Vereb, Csaba, Tóth, Péter, Gergely, László, Nagy, György, Balla, József, Balla
Publikováno v:
Antioxidantsredox signaling. 35(12)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::8d866fd55a94ec4f804d7b0a323f3c88
https://pubmed.ncbi.nlm.nih.gov/34269613
https://pubmed.ncbi.nlm.nih.gov/34269613
Autor:
László Potor, Sikura Katalin Éva, Hajnalka Hegedűs, Dávid Pethő, Zsuzsa Szabó, Zsuzsa M Szigeti, István Pócsi, György Trencsényi, Dezső Szikra, Ildikó Garai, Tamás Gáll, Zsolt Combi, János Kappelmayer György Balla, József Balla
Publikováno v:
International Journal of Molecular Sciences
Volume 21
Issue 13
International Journal of Molecular Sciences, Vol 21, Iss 4746, p 4746 (2020)
Volume 21
Issue 13
International Journal of Molecular Sciences, Vol 21, Iss 4746, p 4746 (2020)
Hemoglobin, heme and iron are implicated in the progression of atherosclerosis. Therefore, we investigated whether the hydrophobic fungal iron chelator siderophore, desferricoprogen (DFC) inhibits atherosclerosis. DFC reduced atherosclerotic plaque f
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=dedup_wf_001::ba2218bf13e289efa633a1cc9bf44538
Autor:
Klára Varga, Roland Veress, Zoltán Balajthy, Ferenc Győry, Abhirup Shaw, Endre Kristóf, Zsolt Bacsó, Zsolt Combi, László Fésüs, Péter Bai, Ágnes Klusóczki
Publikováno v:
Experimental cell research. 377(1-2)
Brown and beige adipocytes contribute significantly to the regulation of whole body energy expenditure and systemic metabolic homeostasis not exclusively by thermogenesis through mitochondrial uncoupling. Several studies have provided evidence in rod
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::39ecacba52c42fcb05e346ceabbec769
https://pubmed.ncbi.nlm.nih.gov/30794803
https://pubmed.ncbi.nlm.nih.gov/30794803