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Akademický článek

Global profiling of protein lactylation in microglia in experimental high-altitude cerebral edema

Autor: Xiufang Jiang, Jiayue Gao, Xuechao Fei, Yanan Geng, Xiangpei Yue, Zibi Shi, Xiang Cheng, Tong Zhao, Ming Fan, Haitao Wu, Ming Zhao, Lingling Zhu

Publikováno v: Cell Communication and Signaling, Vol 22, Iss 1, Pp 1-16 (2024)

Abstract Background High-altitude cerebral edema (HACE) is considered an end-stage acute mountain sickness (AMS) that typically occurs in people after rapid ascent to 2500 m or more. While hypoxia is a fundamental feature of the pathophysiological me

Externí odkaz: https://doaj.org/article/c62b0c9a10f64c1baaaa519c6d733deb

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Akademický článek

Gypenoside-14 Reduces Depression via Downregulation of the Nuclear Factor Kappa B (NF-kB) Signaling Pathway on the Lipopolysaccharide (LPS)-Induced Depression Model

Autor: Yaqun Jiang, Xiang Cheng, Ming Zhao, Tong Zhao, Mengya Zhang, Zibi Shi, Xiangpei Yue, Yanan Geng, Jiayue Gao, Chengbo Wang, Junli Yang, Lingling Zhu

Publikováno v: Pharmaceuticals, Vol 16, Iss 8, p 1152 (2023)

Neuroinflammation is a common pathogenetic sign of depression and is closely linked to the development of depression. Many clinical anti-inflammatory drugs act as antidepressants by reducing the neuroinflammatory response. Previous research found tha

Externí odkaz: https://doaj.org/article/9cd7c256443449ab9b91551e04b11d72

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Cyclooxygenase‑2 contributes to the hypoxia‑induced aggravation of the neuroinflammation response stimulated by lipopolysaccharide in microglia

Autor: Yifan Yang, Yanan Geng, Xiang Cheng, Jiayue Gao, Zibi Shi, Ming Zhao

Publikováno v: Experimental and Therapeutic Medicine. 25

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::2da560c89cac6ed168f43f4579c7c995
https://doi.org/10.3892/etm.2023.11822

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Expression profile of cytokines and chemokines in a mouse high-altitude cerebral edema model

Autor: Zibi Shi, Xiufang Jiang, Yanan Geng, Xiangpei Yue, Jiayue Gao, Xiang Cheng, Ming Zhao, Lingling Zhu

Publikováno v: International Journal of Immunopathology and Pharmacology. 37:039463202311771

Introduction High-altitude cerebral edema (HACE) is considered to be the end-stage of acute mountain sickness (AMS); however, its pathophysiological mechanism remains unknown. Increasing evidences support that inflammation is an important risk factor

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::106f877678c95c99ba2135955e942565
https://doi.org/10.1177/03946320231177189

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