Zobrazeno 1 - 10
of 15
pro vyhledávání: '"Zhuojun, Liao"'
Autor:
Ya Liu, Shiyu Zhou, Jie Qian, Yan Wang, Xiang Yu, Dai Dai, Min Dai, Lingling Wu, Zhuojun Liao, Zhixin Xue, Jiehua Wang, Goujun Hou, Jianyang Ma, John B. Harley, Yuanjia Tang, Nan Shen
Publikováno v:
Arthritis Research & Therapy, Vol 19, Iss 1, Pp 1-11 (2017)
Abstract Background A hallmark of systemic lupus erythematosus is high titers of circulating autoantibodies. Recently, a novel CD11c+ B-cell subset has been identified that is critical for the development of autoimmunity. However, the role of CD11c+
Externí odkaz:
https://doaj.org/article/12053e9f110249988a61bcf9dc199061
Autor:
Zhuojun Liao, Zhizhong Ye, Zhixin Xue, Lingling Wu, Ye Ouyang, Chao Yao, Chaojie Cui, Ning Xu, Jianyang Ma, Guojun Hou, Jiehua Wang, Yao Meng, Zhihua Yin, Ya Liu, Jie Qian, Chunyan Zhang, Huihua Ding, Qiang Guo, Bo Qu, Nan Shen
Publikováno v:
Frontiers in Immunology, Vol 10 (2019)
Objective: Lupus nephritis (LN) is one of the most serious complications of systemic lupus erythematosus (SLE). Type I interferon (IFN-I) is associated with the pathogenesis of LN. Long non-coding RNAs (lncRNAs) have been implicated in the pathogenes
Externí odkaz:
https://doaj.org/article/d123184f94e34039a0699be1e0c77e24
Autor:
Zhixin Xue, Chaojie Cui, Zhuojun Liao, Shiwei Xia, Pingjing Zhang, Jialin Qin, Qiang Guo, Sheng Chen, Qiong Fu, Zhihua Yin, Zhizhong Ye, Yuanjia Tang, Nan Shen
Publikováno v:
Frontiers in Immunology, Vol 9 (2018)
Systemic lupus erythematosus (SLE) is a complex autoimmune disease characterized by augmented type I interferon signaling. High-throughput technologies have identified plenty of SLE susceptibility single-nucleotide polymorphisms (SNPs) yet the exact
Externí odkaz:
https://doaj.org/article/5bb1661b90414536a9c587ccb1d64b33
Publikováno v:
Biochemical and Biophysical Research Communications. 559:141-147
Diabetic nephropathy (DN) is the primary cause of end-stage renal disease, and renal tubular cell dysfunction contributes to the pathogenesis of many kidney diseases. Our previous study demonstrated that dual-specificity protein phosphatase 1 (DUSP1)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3c3d040aafcf555a632abfe74c510492
https://doi.org/10.1016/j.bbrc.2021.04.032
https://doi.org/10.1016/j.bbrc.2021.04.032
Publikováno v:
Toxicology in Vitro. 86:105482
Diabetic nephropathy (DN) is the direct cause of end-stage renal disease, and nephritic inflammation plays a role in its growth and advancement. Aberrant expression of long non-coding RNAs (lncRNAs) correlates with many diseases, including DN. In thi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::faad1435a52a41116fc2da543941e94d
https://doi.org/10.1016/j.tiv.2022.105482
https://doi.org/10.1016/j.tiv.2022.105482
Autor:
Zhihua Yin, Jun Li, Yao Meng, Yuqing Yu, Lu Liu, Haibo Zhou, Guanhua Li, Nan Shen, Li Wu, Zhizhong Ye, Jun Deng, Qing Sun, Zhuojun Liao
Publikováno v:
JCI Insight
Monocyte-derived DCs (moDCs) have been implicated in the pathogenesis of autoimmunity, but the molecular pathways determining the differentiation potential of these cells remain unclear. Here, we report that microRNA-148a (miR-148a) serves as a criti
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4dd122fbfdd22acbf92af0da24d0348d
https://doi.org/10.1172/jci.insight.133721
https://doi.org/10.1172/jci.insight.133721
Autor:
Qiang Guo, Xin Gao, Zhuojun Liao, Jie Qian, Yao Meng, Yange Cui, Nan Shen, Bo Qu, Huihua Ding, Yan Wang, Ya Liu, Lingling Wu, Min Dai, Jiehua Wang, Jun Deng, Chao Yao, Guojun Hou
Publikováno v:
Arthritis & Rheumatology. 70:2036-2045
Objective Increasing evidence indicates that the cyclic GMP-AMP synthase/stimulator of interferon genes (cGAS/STING) signaling pathway has a critical pathogenic role in systemic lupus erythematosus (SLE). Expression levels of the interferon (IFN)-ind
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::e260eb576e9ddb6fcde75e1b1c87684c
https://doi.org/10.1002/art.40576
https://doi.org/10.1002/art.40576
Autor:
Jie Qian, Min Dai, Yuanjia Tang, Dai Dai, John B. Harley, Goujun Hou, Jianyang Ma, Yan Wang, Shiyu Zhou, Xiang Yu, Lingling Wu, Nan Shen, Zhixin Xue, Ya Liu, Jiehua Wang, Zhuojun Liao
Publikováno v:
Arthritis Research & Therapy, Vol 19, Iss 1, Pp 1-11 (2017)
Background A hallmark of systemic lupus erythematosus is high titers of circulating autoantibodies. Recently, a novel CD11c+ B-cell subset has been identified that is critical for the development of autoimmunity. However, the role of CD11c+ B cells i
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::da9ba2b3bb90b1ba034176e67d49b15c
http://link.springer.com/article/10.1186/s13075-017-1438-2
http://link.springer.com/article/10.1186/s13075-017-1438-2
Autor:
Jiehua, Wang, Min, Dai, Yange, Cui, Guojun, Hou, Jun, Deng, Xin, Gao, Zhuojun, Liao, Ya, Liu, Yao, Meng, Lingling, Wu, Chao, Yao, Yan, Wang, Jie, Qian, Qiang, Guo, Huihua, Ding, Bo, Qu, Nan, Shen
Publikováno v:
Arthritisrheumatology (Hoboken, N.J.). 70(12)
Increasing evidence indicates that the cyclic GMP-AMP synthase/stimulator of interferon genes (cGAS/STING) signaling pathway has a critical pathogenic role in systemic lupus erythematosus (SLE). Expression levels of the interferon (IFN)-inducible gen
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::af26d2926dc0a87e6d9fdca74b865a86
https://pubmed.ncbi.nlm.nih.gov/29806091
https://pubmed.ncbi.nlm.nih.gov/29806091
Autor:
Ya, Liu, Shiyu, Zhou, Jie, Qian, Yan, Wang, Xiang, Yu, Dai, Dai, Min, Dai, Lingling, Wu, Zhuojun, Liao, Zhixin, Xue, Jiehua, Wang, Goujun, Hou, Jianyang, Ma, John B, Harley, Yuanjia, Tang, Nan, Shen
Publikováno v:
Arthritis Research & Therapy
Background A hallmark of systemic lupus erythematosus is high titers of circulating autoantibodies. Recently, a novel CD11c+ B-cell subset has been identified that is critical for the development of autoimmunity. However, the role of CD11c+ B cells i
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::2f8c9f4a13a3895b903535e40d5a967f
https://pubmed.ncbi.nlm.nih.gov/28982388
https://pubmed.ncbi.nlm.nih.gov/28982388