Zobrazeno 1 - 10
of 14
pro vyhledávání: '"Zhong-qiang Yao"'
Autor:
Zhong-Qiang Yao1, Hui-Hui Jiang2 jianghh@biotecan.com, Fei-Fei Wang1, Zhi-Gang Fan1, Yi-Ge Zhang1, Shang-Dong Mou1, Xia Cao1, Cheng-Tian Li1, Li-Sha Jiang1, Li Song2, Shu-Shen Ji2, Qing-Juan Chen1 chenqingjuan2023@163.com
Publikováno v:
Journal of Cancer. 2024, Vol. 15 Issue 13, p4205-4218. 14p.
Autor:
Xiuyan Yang, Zhiyi Zhang, Bo Liu, Guochun Wang, Xiangyuan Liu, Ronghua Xie, Danyi Xu, Yunshan Zhou, Yi Liu, Yongfu Wang, Zhanguo Li, Xin Lu, Yan An, Zhuoli Zhang, Hanshi Xu, Xiaoxia Zuo, Yu Wang, Yang Li, Yi Zhao, Lingyun Sun, Yisha Li, Feng Yu, Weiguo Xiao, Jing Zhang, Zhong-qiang Yao, Jianping Guo, Ping Zhu, Hui Shen, Yingnan Li, Yuan An, Ming-Hui Zhao, Xiaoli Deng, Yifang Mei, Hong Wang, Mei Wang, Jin Lin, Liqi Bi
Publikováno v:
Clinical Rheumatology. 38:1047-1054
The standard strategy for treating lupus nephritis comprises glucocorticoids together with either intravenous cyclophosphamide or oral mycophenolate mofetil, but the low remission rate is still a challenge in practice. This study was aimed to seek hi
Autor:
Jinxia Zhao, Xiangyuan Liu, Leilei Jian, Zhenzhen Ma, Changhong Li, Zhong-qiang Yao, Lin Sun, Xinyu Wang
Publikováno v:
International immunopharmacology. 90
Background Iguratimod, a small molecular drug, has been proven to have effective bone protection for treatment of patients with bone loss-related diseases, such as rheumatoid arthritis (RA). However, the exact bone protective mechanism of iguratimod
Autor:
Zhong-qiang Yao, Lin Yang, Xiangyuan Liu, Xiaoli Deng, Rui Xing, Jinxia Zhao, Rui Liu, Lin Sun, Lin-lin Xu, Changhong Li
Publikováno v:
Inflammation Research. 65:193-202
To explore the influence of chemokine, CXCL16, on the expression of the receptor activator nuclear factor κB ligand (RANKL) in rheumatoid arthritis (RA) fibroblast-like synoviocytes (RA-FLS).The expression of CXCL16/CXCR6 and RANKL in RA or osteoart
Publikováno v:
Case Reports in Medicine
Case Reports in Medicine, Vol 2016 (2016)
Case Reports in Medicine, Vol 2016 (2016)
The X-linked hyperimmunoglobulin M syndrome (HIGM), caused by mutations in the CD40LG gene, is a kind of primary immunodeficiency disease (PID). Patients with X-linked HIGM are susceptible to infection as well as autoimmune diseases. Lipoma arboresce
Autor:
Chang-hong, Li, Lin, Sun, Ying-jian, Zhang, Jin-xia, Zhao, Zhong-qiang, Yao, Ning, Xu, Rui, Liu, Xiang-yuan, Liu
Publikováno v:
Beijing da xue xue bao. Yi xue ban = Journal of Peking University. Health sciences. 45(3)
To investigate the expression of Galectins in umbilical cord mesenchymal stem cells (UC-MSCs) from Wharton's jelly.Umbilical cords were obtained sterilely from full term caesarean infants, then mesenchymal stem cells (MSCs) were isolated from Wharton
Publikováno v:
Biochemical and biophysical research communications. 435(4)
Commonly, JAK/STAT relays cytokine signals for cell activation and proliferation, and recent studies have shown that the elevated expression of JAK/STAT is associated with the immune rejection of allografts and the inflammatory processes of autoimmun
Publikováno v:
Biochemical and biophysical research communications. 429(3-4)
The receptor activator of nuclear factor-κB ligand (RANKL), as a member of the tumor necrosis factor (TNF) family, plays an essential role in osteoclast differentiation and function. Chemokines and their receptors have recently been shown to play cr
Autor:
Bujian Tang, Kun Gao, Li Li, Wei Zhang, Xin-Qiang Liang, Qingdi Quentin Li, Yuan Luo, Zhong-Qiang Yao, Zhong-mian Pan
Publikováno v:
Oncology letters. 3(1)
The present study aimed to assess the impact of post-surgical hormone replacement therapy (HRT) on life quality and prognosis in women with ovarian malignancy. HRT (Premarin, Nilestriol and medroxyprogesterone) was administered following surgery in 3
Publikováno v:
Sheng li ke xue jin zhan [Progress in physiology]. 42(1)
Ankylosing spondylitis (AS) is an autoimmune disease which has a strong association with HLA-B27. Its pathogenesis includes the interaction between microorganism and host, the recognition of MHC class I molecule by immune cells and the imbalance of t