Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Zhi-Yang Qiu"'
Publikováno v:
Kaohsiung Journal of Medical Sciences, Vol 40, Iss 5, Pp 422-434 (2024)
Abstract Diabetic foot ulcer (DFU) is a serious complication of diabetic patients which negatively affects their foot health. This study aimed to estimate the role and mechanism of the miR‐200 family in DNA damage of diabetic wound healing. Human f
Externí odkaz:
https://doaj.org/article/302a92abfe554a178335aac411325177
Publikováno v:
Journal of Diabetes Investigation, Vol 14, Iss 12, Pp 1344-1355 (2023)
ABSTRACT Background Umbilical cord‐derived mesenchymal stem cells (UCMSCs) could alleviate diabetes‐induced injury. Hence, this investigation aimed to explore the role and mechanism of UCMSCs‐derived exosomal circHIPK3 (exo‐circHIPK3) in diab
Externí odkaz:
https://doaj.org/article/ee3c0ae3bb3742abbabd356081f75e3b
Autor:
Zun-Hong Liang, Nan-Fang Pan, Shi-Shuai Lin, Zhi-Yang Qiu, Ping Liang, Jun Wang, Zhi Zhang, Yun-Chuan Pan
Publikováno v:
Stem Cell Research & Therapy, Vol 13, Iss 1, Pp 1-13 (2022)
Abstract Background Diabetic foot ulcer (DFU) is a chronic infectious disease caused by diabetes mellitus (DM). Angiogenesis plays the decisive role in wound healing of DFU. Adipose-derived stem cells (ADSCs) can ameliorate angiogenesis in DFU by exo
Externí odkaz:
https://doaj.org/article/46e1d0d2bbaa43ad8ef6dbe774b8badf
Publikováno v:
Regenerative Therapy, Vol 15, Iss , Pp 202-209 (2020)
Background: The process of wound healing is complex. Increasing evidences have shown that lncRNA MALAT1 is abundant in fibroblasts and may be engaged in wound healing process. Therefore, we explored the mechanism of MALAT1 affecting wound healing. Me
Externí odkaz:
https://doaj.org/article/06900534427b4c62a05f7f588484efdc
Autor:
Zun‐Hong Liang, Shi‐Shuai Lin, Nan‐Fang Pan, Guo‐Yu Zhong, Zhi‐Yang Qiu, Shao‐Jia Kuang, Zhi‐Hu Lin, Zhi Zhang, Yun‐Chuan Pan
Publikováno v:
Diabetic medicine : a journal of the British Diabetic AssociationREFERENCES.
Experiments confirmed that circular RNAs contributed to the pathogenesis of diabetic foot ulcers (DFUs). CircHIPK3 was upregulated in type 2 diabetes mellitus (T2DM), but its role in DFU remained unknown. Our study aimed to investigate the regulatory
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::999511432c281d40e31ae8cfd7ec2ba5
https://pubmed.ncbi.nlm.nih.gov/36209373
https://pubmed.ncbi.nlm.nih.gov/36209373
Publikováno v:
Regenerative Therapy, Vol 15, Iss, Pp 202-209 (2020)
Regenerative Therapy
Regenerative Therapy
Background The process of wound healing is complex. Increasing evidences have shown that lncRNA MALAT1 is abundant in fibroblasts and may be engaged in wound healing process. Therefore, we explored the mechanism of MALAT1 affecting wound healing. Met
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5e07d07d60e1ff125e130657faa8903f
https://doi.org/10.1016/j.reth.2020.09.006
https://doi.org/10.1016/j.reth.2020.09.006
Autor:
Zun-Hong Liang, Nan-Fang Pan, Shi-Shuai Lin, Zhi-Yang Qiu, Ping Liang, Jun Wang, Zhi Zhang, Yun-Chuan Pan
Publikováno v:
Stem cell researchtherapy. 13(1)
Background Diabetic foot ulcer (DFU) is a chronic infectious disease caused by diabetes mellitus (DM). Angiogenesis plays the decisive role in wound healing of DFU. Adipose-derived stem cells (ADSCs) can ameliorate angiogenesis in DFU by exosomes. Th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::942dc9ed171b2f0dcf169eb6b5fd0e97
https://pubmed.ncbi.nlm.nih.gov/35870977
https://pubmed.ncbi.nlm.nih.gov/35870977
Publikováno v:
Molecular and cellular biochemistry.
Keloid is a common dermis tumor, occurring repeatedly, affecting the quality of patients' life. Long non-coding RNAs (lncRNAs) have crucial regulatory capacities in skin scarring formation and subsequent scar carcinogenesis. The intention of this stu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::815518fd57974ae6de54c74816dbe92c
https://pubmed.ncbi.nlm.nih.gov/36036334
https://pubmed.ncbi.nlm.nih.gov/36036334