Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Zhi de Cheng"'
Publikováno v:
Proceedings of the 2nd International Conference on Biomedical and Biological Engineering 2017 (BBE 2017).
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::1faa123ac95e9b7494099d4b71a961d9
https://doi.org/10.2991/bbe-17.2017.68
https://doi.org/10.2991/bbe-17.2017.68
Autor:
Ji nan Li, Hai mou Zhang, Dong-xu Liu, Xiang jing Qin, Dong Hua, Yong Chen, Meng yuan Liu, Gang jian Qin, Xiao feng Han, Zhi de Cheng, Gang Liang
Publikováno v:
Molecular Immunology. 46:2876-2884
A major cause of tumor treatment failure is cancer cell metastasis. Toll-like receptor 4 (TLR4)-mediated signaling has been implicated in tumor cell invasion, survival, and metastasis in a variety of cancers. In this study, we investigated the biolog
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::da32ac54182752d6aeeaef6dd4ac3b1e
https://doi.org/10.1016/j.molimm.2009.06.016
https://doi.org/10.1016/j.molimm.2009.06.016
Publikováno v:
Molecular Immunology. 45:1743-1751
C1 inhibitor (C1INH), a member of the serine proteinase inhibitor (serpin) family, functions as an inhibitor of the complement and contact systems. Cleavage of the reactive center loop (RCL) within the carboxyl-terminal domain of C1INH (iC1INH), lack
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bceabee7b75c1ae98ef54d478feb41ad
https://doi.org/10.1016/j.molimm.2007.09.035
https://doi.org/10.1016/j.molimm.2007.09.035
Autor:
Shi-ming Zhu, Xiang-jing Qin, Zi-ming Liu, Zhi-de Cheng, Hai-mou Zhang, Dong-xu Liu, Mengyuan Liu
Publikováno v:
Molecular immunology. 47(6)
Endothelial barrier dysfunction leading to increased permeability and vascular leakage is an underlying cause of several pathological conditions. Whereas these changes have been shown to be associated with activation of the complement system, leading
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d0a300f96edf16d76f4ce022220f92dd
https://pubmed.ncbi.nlm.nih.gov/20138669
https://pubmed.ncbi.nlm.nih.gov/20138669
Autor:
Xin Tu, Qian Liu, Jing Wan, Mengyuan Liu, Gao Chen, Hai-mou Zhang, Hao Xia, Dong-xu Liu, Zhi-de Cheng, Yanxia Wu, Hao Huang, Jinrong Fu, Guosheng Lin
Publikováno v:
Life sciences. 81(17-18)
Acute myocardial infarction (AMI) is associated with inflammation and apoptosis. Emodin plays an anti-inflammatory role in several inflammatory diseases. Recent studies have demonstrated that emodin protects against myocardial ischemia/reperfusion in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4a8375030e7cfb3432beb58fa0174f62
https://pubmed.ncbi.nlm.nih.gov/17939930
https://pubmed.ncbi.nlm.nih.gov/17939930