Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Zhanna Kovalova"'
Autor:
Ieva Nokalna, Madara Kreile, Dagnija Butane, Zhanna Kovalova, Zanda Daneberga, Edvins Miklasevics, Dace Gardovska, Linda Gailite
Publikováno v:
Case Reports in Medicine, Vol 2020 (2020)
X-linked lymphoproliferative disease (XLP) is a rare primary immunodeficiency. Affected individuals usually present with the Epstein–Barr virus infection and have no apparent disease prior to presentation. The most common clinical manifestations ar
Externí odkaz:
https://doaj.org/article/16ac6b382db74baeb4679d911066edcf
Autor:
Petra Ovesná, Mária Kardos, Predrag Miljic, Ernest Bilić, Jan Blatný, Zhanna Kovalova, Majda Benedik-Dolničar, Barbara Faganel-Kotnik, Dobrin Konstantinov
Publikováno v:
Thrombosis research. 198
Introduction This study analyses real-world data on 144 previously untreated patients (PUPs) with severe Haemophilia A, from seven countries in Central and Eastern Europe (CEE: Bulgaria, Croatia, Czech Republic, Hungary, Latvia, Serbia, and Slovenia)
Autor:
Madara Kreile, Linda Gailite, Edvins Miklasevics, Ieva Nokalna, Dace Gardovska, Zanda Daneberga, Zhanna Kovalova, Dagnija Butane
Publikováno v:
Case Reports in Medicine
Case Reports in Medicine, Vol 2020 (2020)
Case Reports in Medicine, Vol 2020 (2020)
X-linked lymphoproliferative disease (XLP) is a rare primary immunodeficiency. Affected individuals usually present with the Epstein–Barr virus infection and have no apparent disease prior to presentation. The most common clinical manifestations ar
Autor:
Barbara De Moerloose, Ninna Bager, Zhanna Kovalova, Berna Beverloo, Bernward Zeller, Eigil Kjeldsen, Julie Damgaard Sandahl, Birgitte Lausen, Olafur G. Jonsson, Eveline S. J. M. de Bont, Kirsi Jahnukainen, Henrik Hasle, Gertjan L. Kaspers, Kadri Saks, Josefine Palle, Ulrika Norén-Nyström, Kristian Løvvik Juul-Dam, Shau-Yin Ha, Jonas Abrahamsson
Publikováno v:
British Journal of Haematology, 183(4), 618-628. Wiley-Blackwell Publishing Ltd
Bager, N, Juul-Dam, K L, Sandahl, J D, Abrahamsson, J, Beverloo, B, de Bont, E S J M, Ha, S-Y, Jahnukainen, K, Jónsson, Ó G, Kaspers, G L, Kovalova, Z, Lausen, B, de Moerloose, B, Noren-Nyström, U, Palle, J, Saks, K, Zeller, B, Kjeldsen, E & Hasle, H 2018, ' Complex and monosomal karyotype are distinct cytogenetic entities with an adverse prognostic impact in paediatric acute myeloid leukaemia. A NOPHO-DBH-AML study ', British Journal of Haematology, vol. 183, no. 4, pp. 618-628 . https://doi.org/10.1111/bjh.15587
Bager, N, Juul-Dam, K L, Sandahl, J D, Abrahamsson, J, Beverloo, B, de Bont, E S J M, Ha, S-Y, Jahnukainen, K, Jónsson, Ó G, Kaspers, G L, Kovalova, Z, Lausen, B, De Moerloose, B, Noren-Nyström, U, Palle, J, Saks, K, Zeller, B, Kjeldsen, E & Hasle, H 2018, ' Complex and monosomal karyotype are distinct cytogenetic entities with an adverse prognostic impact in paediatric acute myeloid leukaemia. A NOPHO-DBH-AML study ', British Journal of Haematology, vol. 183, no. 4, pp. 618-628 . https://doi.org/10.1111/bjh.15587
British Journal of Haematology, 183(4), 618-628. Wiley
British Journal of Haematology, 183(4), 618-628. Wiley-Blackwell
Bager, N, Juul-Dam, K L, Sandahl, J D, Abrahamsson, J, Beverloo, B, de Bont, E S J M, Ha, S-Y, Jahnukainen, K, Jónsson, Ó G, Kaspers, G L, Kovalova, Z, Lausen, B, de Moerloose, B, Noren-Nyström, U, Palle, J, Saks, K, Zeller, B, Kjeldsen, E & Hasle, H 2018, ' Complex and monosomal karyotype are distinct cytogenetic entities with an adverse prognostic impact in paediatric acute myeloid leukaemia. A NOPHO-DBH-AML study ', British Journal of Haematology, vol. 183, no. 4, pp. 618-628 . https://doi.org/10.1111/bjh.15587
Bager, N, Juul-Dam, K L, Sandahl, J D, Abrahamsson, J, Beverloo, B, de Bont, E S J M, Ha, S-Y, Jahnukainen, K, Jónsson, Ó G, Kaspers, G L, Kovalova, Z, Lausen, B, De Moerloose, B, Noren-Nyström, U, Palle, J, Saks, K, Zeller, B, Kjeldsen, E & Hasle, H 2018, ' Complex and monosomal karyotype are distinct cytogenetic entities with an adverse prognostic impact in paediatric acute myeloid leukaemia. A NOPHO-DBH-AML study ', British Journal of Haematology, vol. 183, no. 4, pp. 618-628 . https://doi.org/10.1111/bjh.15587
British Journal of Haematology, 183(4), 618-628. Wiley
British Journal of Haematology, 183(4), 618-628. Wiley-Blackwell
Data on occurrence, genetic characteristics and prognostic impact of complex and monosomal karyotype (CK/MK) in children with acute myeloid leukaemia (AML) are scarce. We studied CK and MK in a large unselected cohort of childhood AML patients diagno
Publikováno v:
Pediatric hematology and oncology. 35(1)
Acute lymphoblastic leukemia (ALL) is the most common childhood malignancy. Modern treatment protocols allow achievement of long-term event-free survival rates in up to 85% of cases, although the treatment response varies among different patient grou
Publikováno v:
Archives of Medical Science : AMS
Introduction Childhood acute lymphoblastic leukaemia (ALL) is a complex disease caused by a combination of genetic susceptibility and environmental exposure. Previous genome-wide association studies have reported several single nucleotide polymorphis
Autor:
Zhanna Kovalova, Madara Kreile, Linda Piekuse, Dmitrijs Rots, Elizabete Cebura, Baiba Lace, Marika Grutupa
Publikováno v:
Asian Pacific Journal of Cancer Prevention. 15:9707-9711
Background: Acute lymphoblastic leukemia (ALL) is a complex disease caused by interactions between hazardous exogenous or/and endogenous agents and many mild effect inherited susceptibility mutations. Some of them are known, but their functional role
Autor:
Gunta Chernevska, Zhanna Kovalova, Arturs Sochnevs, Jelena Eglite, Velga Kuse, Sandra Lejniece, Raimonds Simanis, Dace Gardovska, Agita Jeruma, Ludmila Viksna
Publikováno v:
Medicina; Volume 44; Issue 1; Pages: 15
Scopus-Elsevier
Medicina
Volume 44
Issue 1
Scopus-Elsevier
Medicina
Volume 44
Issue 1
Objective. The objective of this study was to investigate the prevalence of HCV (hepatitis C virus) infection in hemophilia patients in Latvia and to analyze association between natural clearance of HCV and human leukocyte antigen (HLA) class II gene