Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Zeliha Gozde Turan"'
Autor:
Ester Kalef-Ezra, Zeliha Gozde Turan, Diego Perez-Rodriguez, Ida Bomann, Sairam Behera, Caoimhe Morley, Sonja W. Scholz, Zane Jaunmuktane, Jonas Demeulemeester, Fritz J. Sedlazeck, Christos Proukakis
Publikováno v:
Communications Biology, Vol 7, Iss 1, Pp 1-10 (2024)
Abstract The presence of somatic mutations, including copy number variants (CNVs), in the brain is well recognized. Comprehensive study requires single-cell whole genome amplification, with several methods available, prior to sequencing. Here we comp
Externí odkaz:
https://doaj.org/article/33142598378f4a9a88091ba6a21379f4
Autor:
Poorya Parvizi, Handan Melike Dönertaş, Jenny Tung, Zeliha Gozde Turan, Philipp Khaitovich, Mehmet Somel
Publikováno v:
Aging Cell
Turan, Z G, Parvizi, P, Dönertaş, H M, Tung, J, Khaitovich, P & Somel, M 2019, ' Molecular footprint of Medawar's mutation accumulation process in mammalian aging ', Aging Cell, pp. e12965 . https://doi.org/10.1111/acel.12965
Turan, Z G, Parvizi, P, Dönertaş, H M, Tung, J, Khaitovich, P & Somel, M 2019, ' Molecular footprint of Medawar's mutation accumulation process in mammalian aging ', Aging Cell, pp. e12965 . https://doi.org/10.1111/acel.12965
Medawar's mutation accumulation hypothesis explains aging by the declining force of natural selection with age: Slightly deleterious germline mutations expressed in old age can drift to fixation and thereby lead to aging‐related phenotypes. Althoug
Autor:
Handan Melike Dönertaş, Zeliha Gozde Turan, Philipp Khaitovich, Mehmet Somel, Jenny Tung, Poorya Parvizi
Medawar’s mutation accumulation (MA) hypothesis explains ageing by the declining force of natural selection with age: slightly deleterious germline mutations that are functional in old age are not effectively eliminated by selection and therefore l
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::99581a665657f729c2dced84c66f45b5
Autor:
Zeliha Gözde Turan, Vincent Richter, Jana Bochmann, Poorya Parvizi, Etka Yapar, Ulas Işıldak, Sarah-Kristin Waterholter, Sabrina Leclere-Turbant, Çağdaş Devrim Son, Charles Duyckaerts, İdil Yet, Thomas Arendt, Mehmet Somel, Uwe Ueberham
Publikováno v:
Acta Neuropathologica Communications, Vol 10, Iss 1, Pp 1-16 (2022)
Abstract The possible role of somatic copy number variations (CNVs) in Alzheimer’s disease (AD) aetiology has been controversial. Although cytogenetic studies suggested increased CNV loads in AD brains, a recent single-cell whole-genome sequencing
Externí odkaz:
https://doaj.org/article/ba317eb633834c2a8760cce667a8cb1c