Zobrazeno 1 - 10
of 35
pro vyhledávání: '"Zeina Jaffar"'
Publikováno v:
Frontiers in Pharmacology, Vol 9 (2018)
Multi-walled carbon nanotubes (MWCNT) have been reported to promote lung inflammation and fibrosis. The commercial demand for nanoparticle-based materials has expanded rapidly and as demand for nanomaterials grows, so does the urgency of establishing
Externí odkaz:
https://doaj.org/article/e2927be47c864ae79a209381ddae21d9
Autor:
Maria E Ferrini, Bryan J Simons, David J P Bassett, Matthews O Bradley, Kevan Roberts, Zeina Jaffar
Publikováno v:
PLoS ONE, Vol 8, Iss 7, p e70351 (2013)
Allergic asthma is characterized by Th2 type inflammation, leading to airway hyperresponsivenes, mucus hypersecretion and tissue remodeling. S-Nitrosoglutathione reductase (GSNOR) is an alcohol dehydrogenase involved in the regulation of intracellula
Externí odkaz:
https://doaj.org/article/c52d09b814cb4b65a98008ffbd2ef4cb
Autor:
Yang Jee Kim, Yoon Hee Cho, Kent E. Pinkerton, Virginia Porter, Mihi Yang, Sonja Christensen, Andrij Holian, Maria Ferrini, Elizabeth Cole, Zeina Jaffar, Luke Montrose, Britten Postma, Kevan Roberts
Publikováno v:
Environmental and Molecular Mutagenesis. 58:423-433
Allergic asthma remains an inadequately understood disease. In utero exposure to environmental tobacco smoke (ETS) has been identified as an environmental exposure that can increase an individual's asthma risk. To improve our understanding of asthma
Autor:
Zeina Jaffar, N. Stella, Maria Ferrini, Donald B. Stierle, Kevan Roberts, S. Hong, Andrea A. Stierle
Publikováno v:
Allergy. 72:937-947
Background Allergic asthma is a chronic airway inflammatory disease involving the complementary actions of innate and adaptive immune responses. Endogenously generated cannabinoids acting via CB2 receptors play important roles in both homeostatic and
Autor:
Kevan Roberts, Bryan J. Simons, Zeina Jaffar, Sophia Carvalho, Maria Ferrini, David J. P. Bassett
Publikováno v:
The Journal of Immunology. 198:461-471
In allergic asthma, inhalation of airborne allergens such as the house dust mite (HDM) effectively activates both innate and adaptive immunity in the lung mucosa. To determine the role of the eicosanoid PGI2 and its receptor IP during allergic airway
Publikováno v:
The Journal of Immunology. 204:147.5-147.5
The alarmin, IL-33 plays a critical role in the inception of lung inflammation and the exacerbation of allergic airway disease. Inhalation of environmental allergens and particulates is known to promote IL-33 release, and the cells responsive to this
Publikováno v:
Frontiers in Pharmacology, Vol 9 (2018)
Multi-walled carbon nanotubes (MWCNT) have been reported to promote lung inflammation and fibrosis. The commercial demand for nanoparticle-based materials has expanded rapidly and as demand for nanomaterials grows, so does the urgency of establishing
Autor:
Zeina Jaffar, Maria Ferrini, Sophia Carvalho, Britten Postma, Kent E. Pinkerton, Kevan Roberts, Yoon Hee Cho, Lucas Miranda Marques
Publikováno v:
Particle and Fibre Toxicology
Particle and fibre toxicology, vol 14, iss 1
Particle and fibre toxicology, vol 14, iss 1
Background Epidemiological studies suggest that prenatal and early life environmental exposures have adverse effects on pulmonary function and are important contributors in the development of childhood asthma and allergic disease. The mechanism by wh
Publikováno v:
The Journal of Immunology. 202:119.28-119.28
The alarmin, IL-33, acting via ST2 receptors initiates innate immunity to inhaled allergens and environmental particulates. IL-33 belongs to the IL-1 family and is largely derived from epithelial cells, endothelial cells and fibroblasts. The cytokine
Autor:
Sonja, Christensen, Zeina, Jaffar, Elizabeth, Cole, Virginia, Porter, Maria, Ferrini, Britten, Postma, Kent E, Pinkerton, Mihi, Yang, Yang Jee, Kim, Luke, Montrose, Kevan, Roberts, Andrij, Holian, Yoon Hee, Cho
Publikováno v:
Environmental and molecular mutagenesis. 58(6)
Allergic asthma remains an inadequately understood disease. In utero exposure to environmental tobacco smoke (ETS) has been identified as an environmental exposure that can increase an individual's asthma risk. To improve our understanding of asthma