Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Zdena Polivkova"'
Autor:
Veronika Vymetalkova, Pavel Soucek, Tereza Kunicka, Katerina Jiraskova, Veronika Brynychova, Barbara Pardini, Vendula Novosadova, Zdena Polivkova, Katerina Kubackova, Renata Kozevnikovova, Miloslav Ambrus, Ludmila Vodickova, Alessio Naccarati, Pavel Vodicka
Publikováno v:
PLoS ONE, Vol 10, Iss 7, p e0134463 (2015)
Variations in the TP53 gene have been suggested to play a role in many cancers, including breast. We previously observed an association between TP53 haplotypes based on four polymorphisms (rs17878362, rs1042522, rs12947788, and rs17884306) and the ri
Externí odkaz:
https://doaj.org/article/daa9818343444ee98abf92c5511e42d0
Autor:
Asta Försti, Sona Vodenkova, Pavel Vodicka, Ludmila Vodickova, Zdena Polivkova, Kari Hemminki, Ludovit Musak, Alessio Naccarati, Calogerina Catalano, Veronika Vymetalkova, Michal Kroupa
Publikováno v:
Mutation research. Genetic toxicology and environmental mutagenesis. 836
Human malignancies are often hallmarked with genomic instability, which itself is also considered a causative event in malignant transformation. Genomic instability may manifest itself as genetic changes in the nucleotide sequence of DNA, or as struc
Autor:
Tereza Kunická, Pavel Soucek, Ludmila Vodickova, Pavel Vodicka, Zdena Polivkova, Veronika Vymetalkova, Miloslav Ambrus, Veronika Brynychova, Katerina Kubackova, Vendula Novosadova, Renata Kozevnikovova, Alessio Naccarati, Katerina Jiraskova, Barbara Pardini
Publikováno v:
PLoS ONE, Vol 10, Iss 7, p e0134463 (2015)
PLoS ONE
PLoS ONE
Variations in the TP53 gene have been suggested to play a role in many cancers, including breast. We previously observed an association between TP53 haplotypes based on four polymorphisms (rs17878362, rs1042522, rs12947788, and rs17884306) and the ri
Autor:
Alessio Naccarati, Ludmila Vodickova, Zdena Polivkova, Michal Kroupa, Rajesh Kumar, Ludovit Musak, Kari Hemminki, Maria Dusinska, Veronika Vymetalkova, Pavel Vodicka, Sona Vodenkova
Publikováno v:
Cancer Research. 76:801-801
Human cancers arise from cells unable to maintain genomic and chromosomal stability, mainly as a sequential consequence of altered DNA repair mechanisms (base and nucleotide excision, mismatch and double-strand breaks). Chromosomal aberrations (CAs)