Zobrazeno 1 - 10
of 19
pro vyhledávání: '"Zanda Bakaeva"'
Autor:
Zanda Bakaeva, Natalia Lizunova, Ivan Tarzhanov, Dmitrii Boyarkin, Svetlana Petrichuk, Vsevolod Pinelis, Andrey Fisenko, Alexander Tuzikov, Rinat Sharipov, Alexander Surin
Publikováno v:
Frontiers in Molecular Neuroscience, Vol 14 (2022)
Lipopolysaccharide (LPS), a fragment of the bacterial cell wall, specifically interacting with protein complexes on the cell surface, can induce the production of pro-inflammatory and apoptotic signaling molecules, leading to the damage and death of
Externí odkaz:
https://doaj.org/article/ec06637c904e4ab19fd84deb55ff3503
Publikováno v:
Molecular Brain, Vol 12, Iss 1, Pp 1-4 (2019)
Abstract Aim An impaired biological response to insulin in the brain, known as central insulin resistance, was identified during stroke and traumatic brain injury, for which glutamate excitotoxicity is a common pathogenic factor. The exact molecular
Externí odkaz:
https://doaj.org/article/caa7739e3eac4798948fd603d0c43234
Autor:
Arina Zgodova, Svetlana Pavlova, Anastasia Nekrasova, Dmitriy Boyarkin, Vsevolod Pinelis, Alexander Surin, Zanda Bakaeva
Publikováno v:
Membranes, Vol 12, Iss 11, p 1052 (2022)
It is considered that glutamate excitotoxicity may be a major factor in the pathological death of neurons and mediate the development of neurodegenerative diseases in humans. Here, we show that isoliquiritigenin (ILG) at a concentration of 0.5–5 µ
Externí odkaz:
https://doaj.org/article/34546ec7114d4c6fa46781a6d9fc9ea5
Autor:
Vsevolod Pinelis, Irina Krasilnikova, Zanda Bakaeva, Alexander Surin, Dmitrii Boyarkin, Andrei Fisenko, Olga Krasilnikova, Igor Pomytkin
Publikováno v:
International Journal of Molecular Sciences; Volume 23; Issue 20; Pages: 12593
Glutamate excitotoxicity is involved in the pathogenesis of many disorders, including stroke, traumatic brain injury, and Alzheimer’s disease, for which central insulin resistance is a comorbid condition. Neurotoxicity of glutamate (Glu) is primari
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::07b9ca4197e1127ac7d213a1f42eb7c2
https://pubmed.ncbi.nlm.nih.gov/36293449
https://pubmed.ncbi.nlm.nih.gov/36293449
Publikováno v:
Molecular Brain, Vol 12, Iss 1, Pp 1-4 (2019)
Molecular Brain
Molecular Brain
AimAn impaired biological response to insulin in the brain, known as central insulin resistance, was identified during stroke and traumatic brain injury, for which glutamate excitotoxicity is a common pathogenic factor. The exact molecular link betwe
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1c044b2b08907341d2477fda6c94dfd8
https://doaj.org/article/caa7739e3eac4798948fd603d0c43234
https://doaj.org/article/caa7739e3eac4798948fd603d0c43234
Autor:
Zanda, Bakaeva, Natalia, Lizunova, Ivan, Tarzhanov, Dmitrii, Boyarkin, Svetlana, Petrichuk, Vsevolod, Pinelis, Andrey, Fisenko, Alexander, Tuzikov, Rinat, Sharipov, Alexander, Surin
Publikováno v:
Frontiers in Molecular Neuroscience
Lipopolysaccharide (LPS), a fragment of the bacterial cell wall, specifically interacting with protein complexes on the cell surface, can induce the production of pro-inflammatory and apoptotic signaling molecules, leading to the damage and death of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::8b37116b659c100d07431d911fb73450
https://pubmed.ncbi.nlm.nih.gov/35069113
https://pubmed.ncbi.nlm.nih.gov/35069113
Autor:
Olga A. Krasilnikova, Zanda Bakaeva, Dmitry Boyarkin, Vsevolod Pinelis, Alexander Surin, Andrey P. Fisenko, Igor Pomytkin, Irina Krasil’nikova
Glutamate excitotoxicity is implicated in the pathogenesis of many disorders, including stroke, traumatic brain injury, and Alzheimer’s disease, for which central insulin resistance is a comorbid condition. Massive glutamate release primarily throu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d0005351fdba41885b02a87b7c5dcac5
Autor:
Zanda Bakaeva, Mikhail Goncharov, Irina Krasilnikova, Arina Zgodova, Daniil Frolov, Ekaterina Grebenik, Peter Timashev, Vsevolod Pinelis, Alexander Surin
Publikováno v:
International Journal of Molecular Sciences; Volume 23; Issue 7; Pages: 3858
In vitro models of traumatic brain injury (TBI) help to elucidate the pathological mechanisms responsible for cell dysfunction and death. To simulate in vitro the mechanical brain trauma, primary neuroglial cultures were scratched during different pe
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ab1fe664778c51c5c98070ccec42da63
https://doi.org/10.3390/ijms23073858
https://doi.org/10.3390/ijms23073858
Autor:
Konstantin N. Belosludtsev, Irina Krasil’nikova, Zanda Bakaeva, Natalia V. Belosludtseva, Arina E. Zgodova, Dmitry Boyarkin, Mikhail V. Dubinin, Rinat R. Sharipov, Alexander Surin, Vsevolod Pinelis
Publikováno v:
Biochimica et biophysica acta. General subjects. 1865(5)
Background Disorders of mitochondrial Ca2+ homeostasis play a key role in the glutamate excitotoxicity of brain neurons. DS16570511 (DS) is a new penetrating inhibitor of mitochondrial Ca2+ uniporter complex (MCUC). The paper examines the effects of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::66d5d219f09ee8a8fdfd06e19b9d6a31
https://pubmed.ncbi.nlm.nih.gov/33453305
https://pubmed.ncbi.nlm.nih.gov/33453305
Viscoelasticity and Volume of Cortical Neurons under Glutamate Excitotoxicity and Osmotic Challenges
Autor:
Zanda Bakaeva, Ekaterina A. Grebenik, Irina Krasil’nikova, Alexander Surin, A. A. Akovantseva, Rinat R. Sharipov, Yuri M. Efremov, Pinelis Vg, Peter S. Timashev, Svetlana L. Kotova
Publikováno v:
Biophys J
Neural activity depends on the maintenance of ionic and osmotic homeostasis. Under these conditions, the cell volume must be regulated to maintain optimal neural function. A disturbance in the neuronal volume regulation often occurs in pathological c
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8a7122568b46953804a6f681991c93f3
https://pubmed.ncbi.nlm.nih.gov/33086042
https://pubmed.ncbi.nlm.nih.gov/33086042