Palmitoylethanolamide inhibits the expression of fatty acid amide hydrolase and enhances the anti-proliferative effect of anandamide in human breast cancer cells

Autor: Di Marzo V., Melck D., Orlando P., Bisogno T., Zagoory O., Bifulco M., Vogel Z., De Petrocellis L.

Publikováno v: Biochemical journal (Lond., 1984) 358 (2001): 249–255. doi:10.1042/0264-6021:3580249
info:cnr-pdr/source/autori:Di Marzo V., Melck D., Orlando P., Bisogno T., Zagoory O., Bifulco M., Vogel Z., De Petrocellis L./titolo:Palmitoylethanolamide inhibits the expression of fatty acid amide hydrolase and enhances the anti-proliferative effect of anandamide in human breast cancer cells/doi:10.1042%2F0264-6021:3580249/rivista:Biochemical journal (Lond., 1984)/anno:2001/pagina_da:249/pagina_a:255/intervallo_pagine:249–255/volume:358

Palmitoylethanolamide (PEA) has been shown to act in synergy with anandamide (arachidonoylethanolamide; AEA), an endogenous agonist of cannabinoid receptor type 1 (CB(1)). This synergistic effect was reduced by the CB(2) cannabinoid receptor antagoni

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https://pubmed.ncbi.nlm.nih.gov/11485574

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Examining effects of postnatal maternal care on offspring phenotype by "switching" the pups between dams of one line and another.

Autor: Weller, A., Brunelli, S. A., Schroeder, M., Malkesman, O., Lavi-Avnon, Y., Hurwitz, I., Blumberg, S., Zagoory, O.

Publikováno v: Neural Plasticity; 2005, Vol. 12 Issue 1, p63-63, 1/4p