Corrigendum: Persistent upregulation of the β-tubulin tubb6, linked to muscle regeneration, is a source of microtubule disorganization in dystrophic muscle.

Autor: Randazzo D; Light Imaging Section, Office of Science and Technology, National Institute of Arthritis and Musculoskeletal and Skin Diseases (NIAMS), National Institutes of Health (NIH), Bethesda, MD, USA., Khalique U; Light Imaging Section, Office of Science and Technology, National Institute of Arthritis and Musculoskeletal and Skin Diseases (NIAMS), National Institutes of Health (NIH), Bethesda, MD, USA., Belanto JJ; Department of Biochemistry, Molecular Biology, and Biophysics, and Program in Molecular, Cellular, Developmental Biology, and Genetics, University of Minnesota-Twin Cities, Minneapolis, MN, USA., Kenea A; Light Imaging Section, Office of Science and Technology, National Institute of Arthritis and Musculoskeletal and Skin Diseases (NIAMS), National Institutes of Health (NIH), Bethesda, MD, USA., Talsness DM; Department of Biochemistry, Molecular Biology, and Biophysics, and Program in Molecular, Cellular, Developmental Biology, and Genetics, University of Minnesota-Twin Cities, Minneapolis, MN, USA., Olthoff JT; Department of Biochemistry, Molecular Biology, and Biophysics, and Program in Molecular, Cellular, Developmental Biology, and Genetics, University of Minnesota-Twin Cities, Minneapolis, MN, USA., Tran MD; Light Imaging Section, Office of Science and Technology, National Institute of Arthritis and Musculoskeletal and Skin Diseases (NIAMS), National Institutes of Health (NIH), Bethesda, MD, USA., Zaal KJ; Light Imaging Section, Office of Science and Technology, National Institute of Arthritis and Musculoskeletal and Skin Diseases (NIAMS), National Institutes of Health (NIH), Bethesda, MD, USA., Pak K; Laboratory of Muscle Stem Cells and Gene Regulation, Muscle Disease Unit, NIAMS, NIH, Bethesda, MD, USA., Pinal-Fernandez I; Laboratory of Muscle Stem Cells and Gene Regulation, Muscle Disease Unit, NIAMS, NIH, Bethesda, MD, USA.; Johns Hopkins University School of Medicine, Baltimore, MD, USA., Mammen AL; Laboratory of Muscle Stem Cells and Gene Regulation, Muscle Disease Unit, NIAMS, NIH, Bethesda, MD, USA.; Johns Hopkins University School of Medicine, Baltimore, MD, USA., Sackett D; Eunice Kennedy Shriver National Institute of Child Health and Human Development (NICHD), NIH, Bethesda, MD, USA., Ervasti JM; Department of Biochemistry, Molecular Biology, and Biophysics, and Program in Molecular, Cellular, Developmental Biology, and Genetics, University of Minnesota-Twin Cities, Minneapolis, MN, USA., Ralston E; Light Imaging Section, Office of Science and Technology, National Institute of Arthritis and Musculoskeletal and Skin Diseases (NIAMS), National Institutes of Health (NIH), Bethesda, MD, USA.

Publikováno v: Human molecular genetics [Hum Mol Genet] 2019 Jun 01; Vol. 28 (11), pp. 1930.

β-Actin shows limited mobility and is required only for supraphysiological insulin-stimulated glucose transport in young adult soleus muscle.

Autor: Madsen AB; Department of Nutrition, Exercise and Sports, University of Copenhagen , Copenhagen , Denmark., Knudsen JR; Department of Nutrition, Exercise and Sports, University of Copenhagen , Copenhagen , Denmark., Henriquez-Olguin C; Department of Nutrition, Exercise and Sports, University of Copenhagen , Copenhagen , Denmark.; Centro de Estudios Moleculares de la Célula, Instituto de Ciencias Biomédicas, Universidad de Chile ; Laboratory of Exercise Sciences, Clínica MEDS, Santiago , Chile., Angin Y; Department of Nutrition, Exercise and Sports, University of Copenhagen , Copenhagen , Denmark., Zaal KJ; Light Imaging Section, Office of Science and Technology, National Institute of Arthritis and Musculoskeletal and Skin Diseases, National Institutes of Health , Bethesda, Maryland., Sylow L; Department of Nutrition, Exercise and Sports, University of Copenhagen , Copenhagen , Denmark., Schjerling P; Institute of Sports Medicine, Department of Orthopedic Surgery, Bispebjerg Hospital , Copenhagen , Denmark.; Center of Healthy Aging, Faculty of Health and Medical Sciences, University of Copenhagen , Copenhagen , Denmark., Ralston E; Light Imaging Section, Office of Science and Technology, National Institute of Arthritis and Musculoskeletal and Skin Diseases, National Institutes of Health , Bethesda, Maryland., Jensen TE; Department of Nutrition, Exercise and Sports, University of Copenhagen , Copenhagen , Denmark.

Publikováno v: American journal of physiology. Endocrinology and metabolism [Am J Physiol Endocrinol Metab] 2018 Jul 01; Vol. 315 (1), pp. E110-E125. Date of Electronic Publication: 2018 Mar 13.

An activating NLRC4 inflammasome mutation causes autoinflammation with recurrent macrophage activation syndrome.

Autor: Canna SW; Molecular Immunology and Inflammation Branch, National Institute for Arthritis and Musculoskeletal and Skin Diseases (NIAMS), US National Institutes of Health (NIH), Bethesda, Maryland, USA., de Jesus AA; Translational Autoinflammatory Disease Section, NIAMS, NIH, Bethesda, Maryland, USA., Gouni S; Molecular Immunology and Inflammation Branch, National Institute for Arthritis and Musculoskeletal and Skin Diseases (NIAMS), US National Institutes of Health (NIH), Bethesda, Maryland, USA., Brooks SR; Biodata Mining and Discovery Section, Office of Science and Technology, NIAMS, NIH, Bethesda, Maryland, USA., Marrero B; Translational Autoinflammatory Disease Section, NIAMS, NIH, Bethesda, Maryland, USA., Liu Y; Translational Autoinflammatory Disease Section, NIAMS, NIH, Bethesda, Maryland, USA., DiMattia MA; Laboratory of Structural Biology, NIAMS, NIH, Bethesda, Maryland, USA., Zaal KJ; Light Imaging Section, Office of Science and Technology, NIAMS, NIH, Bethesda, Maryland, USA., Sanchez GA; 1] Translational Autoinflammatory Disease Section, NIAMS, NIH, Bethesda, Maryland, USA. [2] Office of the Clinical Director, NIAMS, NIH, Bethesda, Maryland, USA., Kim H; Translational Autoinflammatory Disease Section, NIAMS, NIH, Bethesda, Maryland, USA., Chapelle D; 1] Translational Autoinflammatory Disease Section, NIAMS, NIH, Bethesda, Maryland, USA. [2] Office of the Clinical Director, NIAMS, NIH, Bethesda, Maryland, USA., Plass N; 1] Translational Autoinflammatory Disease Section, NIAMS, NIH, Bethesda, Maryland, USA. [2] Office of the Clinical Director, NIAMS, NIH, Bethesda, Maryland, USA., Huang Y; Translational Autoinflammatory Disease Section, NIAMS, NIH, Bethesda, Maryland, USA., Villarino AV; Molecular Immunology and Inflammation Branch, National Institute for Arthritis and Musculoskeletal and Skin Diseases (NIAMS), US National Institutes of Health (NIH), Bethesda, Maryland, USA., Biancotto A; Center for Human Immunology, National Heart, Lung, and Blood Institute, NIH, Bethesda, Maryland, USA., Fleisher TA; Department of Laboratory Medicine, NIH Clinical Center, Bethesda, Maryland, USA., Duncan JA; Infectious Diseases, University of North Carolina School of Medicine, Chapel Hill, North Carolina, USA., O'Shea JJ; Molecular Immunology and Inflammation Branch, National Institute for Arthritis and Musculoskeletal and Skin Diseases (NIAMS), US National Institutes of Health (NIH), Bethesda, Maryland, USA., Benseler S; Pediatric Rheumatology, Alberta Children's Hospital and University of Calgary, Calgary, Alberta, Canada., Grom A; Pediatric Rheumatology, Cincinnati Children's Hospital and University of Cincinnati, Cincinnati, Ohio, USA., Deng Z; Biodata Mining and Discovery Section, Office of Science and Technology, NIAMS, NIH, Bethesda, Maryland, USA., Laxer RM; Pediatric Rheumatology, Hospital for Sick Children, Toronto, Ontario, Canada., Goldbach-Mansky R; Translational Autoinflammatory Disease Section, NIAMS, NIH, Bethesda, Maryland, USA.

Publikováno v: Nature genetics [Nat Genet] 2014 Oct; Vol. 46 (10), pp. 1140-6. Date of Electronic Publication: 2014 Sep 14.

Microtubules that form the stationary lattice of muscle fibers are dynamic and nucleated at Golgi elements.

Autor: Oddoux S; Light Imaging Section, Office of Science and Technology, National Institute of Arthritis and Musculoskeletal and Skin Diseases, National Institutes of Health, Bethesda, MD 20892., Zaal KJ, Tate V, Kenea A, Nandkeolyar SA, Reid E, Liu W, Ralston E

Publikováno v: The Journal of cell biology [J Cell Biol] 2013 Oct 28; Vol. 203 (2), pp. 205-13. Date of Electronic Publication: 2013 Oct 21.

Who needs microtubules? Myogenic reorganization of MTOC, Golgi complex and ER exit sites persists despite lack of normal microtubule tracks.

Autor: Zaal KJ; Light Imaging Section, Office of Science and Technology, National Institute of Arthritis, Musculoskeletal, and Skin Disease, National Institutes of Health, Bethesda, Maryland, USA. zaalk@mail.nih.gov, Reid E, Mousavi K, Zhang T, Mehta A, Bugnard E, Sartorelli V, Ralston E

Publikováno v: PloS one [PLoS One] 2011; Vol. 6 (12), pp. e29057. Date of Electronic Publication: 2011 Dec 27.