Zobrazeno 1 - 10
of 96
pro vyhledávání: '"Yutaka Kakizoe"'
Autor:
Yoshikazu Miyasato, Jun Morinaga, Hideki Inoue, Yushi Nakayama, Masataka Adachi, Yuichiro Izumi, Yutaka Kakizoe, Teruhiko Mizumoto, Terumasa Nakagawa, Daisuke Fujimoto, Makoto Ono, Akira Oda, Takashige Kuwabara, Masashi Mukoyama
Publikováno v:
Renal Replacement Therapy, Vol 9, Iss 1, Pp 1-7 (2023)
Abstract Background Malnutrition is associated with discontinuing peritoneal dialysis (PD). The prognostic nutritional index (PNI), composed of serum albumin level and total lymphocyte count, has been suggested as a prognostic marker for mortality in
Externí odkaz:
https://doaj.org/article/4cce6ad4abe34fcf9826b91a2868e057
Autor:
Yutaka Kakizoe, Terumasa Nakagawa, Yasunobu Iwata, Qinyuan Deng, Masataka Adachi, Yoshikazu Miyasato, Miyuki Nakagawa, Yu Nagayoshi, Kayo Nishiguchi, Yuki Narita, Yuichiro Izumi, Takashige Kuwabara, Kimio Tomita, Kenichiro Kitamura, Masashi Mukoyama
Publikováno v:
Journal of Pharmacological Sciences, Vol 150, Iss 4, Pp 204-210 (2022)
Serine proteases (SPs) play physiological roles in the kidney. We previously reported that a synthetic SP inhibitor, camostat mesilate (CM), suppressed sodium reabsorption in the renal tubule and showed natriuretic effects in aldosterone-infused rats
Externí odkaz:
https://doaj.org/article/eecb1b08290043fca9c1a02abeb6e4f3
Autor:
Yasunobu Iwata, Qinyuan Deng, Yutaka Kakizoe, Terumasa Nakagawa, Yoshikazu Miyasato, Miyuki Nakagawa, Kayo Nishiguchi, Yu Nagayoshi, Yuki Narita, Yuichiro Izumi, Takashige Kuwabara, Masataka Adachi, Masashi Mukoyama
Publikováno v:
International Journal of Molecular Sciences, Vol 24, Iss 21, p 15743 (2023)
In proteinuric renal diseases, the serine protease (SP) plasmin activates the epithelial sodium channel (ENaC) by cleaving its γ subunit. We previously demonstrated that a high-salt (HS) diet provoked hypertension and proteinuria in Dahl salt-sensit
Externí odkaz:
https://doaj.org/article/0bd65411bec1419995b0b6e982f0fc6b
Autor:
Teruhiko Mizumoto, Yutaka Kakizoe, Terumasa Nakagawa, Yasunobu Iwata, Yoshikazu Miyasato, Kohei Uchimura, Masataka Adachi, Qinyuan Deng, Manabu Hayata, Jun Morinaga, Taku Miyoshi, Yuichiro Izumi, Takashige Kuwabara, Yoshiki Sakai, Kimio Tomita, Kenichiro Kitamura, Masashi Mukoyama
Publikováno v:
Journal of Pharmacological Sciences, Vol 146, Iss 4, Pp 192-199 (2021)
Metabolic syndrome (MetS) is associated with chronic kidney disease and proteinuria. Previously, we reported that a synthetic serine protease inhibitor, camostat mesilate (CM), mitigated hypertension and proteinuria in rodent disease models. The pres
Externí odkaz:
https://doaj.org/article/59eec7db31444310acc443e68816d53e
Autor:
Huixian Hou, Mai Horikawa, Yuki Narita, Hirofumi Jono, Yutaka Kakizoe, Yuichiro Izumi, Takashige Kuwabara, Masashi Mukoyama, Hideyuki Saito
Publikováno v:
International Journal of Molecular Sciences, Vol 24, Iss 14, p 11329 (2023)
Renal fibrosis is the final manifestation of chronic kidney disease (CKD); its prevention is vital for controlling CKD progression. Indoxyl sulfate (IS), a typical sulfate-conjugated uremic solute, is produced in the liver via the enzyme sulfotransfe
Externí odkaz:
https://doaj.org/article/5d849d265c3c427491a849afb05038d6
Autor:
Tomoko Kanki, Takashige Kuwabara, Jun Morinaga, Hirotaka Fukami, Shuro Umemoto, Daisuke Fujimoto, Teruhiko Mizumoto, Manabu Hayata, Yutaka Kakizoe, Yuichiro Izumi, Saeko Tajiri, Tetsuya Tajiri, Kenichiro Kitamura, Masashi Mukoyama
Publikováno v:
BMC Nephrology, Vol 21, Iss 1, Pp 1-10 (2020)
Abstract Background The inflammatory mediator calprotectin (CPT, myeloid-related protein 8/14) is known as an endogenous ligand contributing to pathophysiology in inflammatory diseases. Serum CPT reportedly became a potential biomarker in these condi
Externí odkaz:
https://doaj.org/article/80d7e54cbc8e450ba80931d97f36d01d
Autor:
Yu Nagayoshi, Kayo Nishiguchi, Ryosuke Yamamura, Takeshi Chujo, Hiroyuki Oshiumi, Hiroko Nagata, Hitomi Kaneko, Keiichi Yamamoto, Hirotomo Nakata, Korin Sakakida, Akihiro Kunisawa, Masataka Adachi, Yutaka Kakizoe, Takanori Mizobe, Jun-ichi Kuratsu, Shinya Shimada, Yasushi Nakamori, Masao Matsuoka, Masashi Mukoyama, Fan-Yan Wei, Kazuhito Tomizawa
Publikováno v:
Biomolecules, Vol 12, Iss 9, p 1233 (2022)
SARS-CoV-2 infection alters cellular RNA content. Cellular RNAs are chemically modified and eventually degraded, depositing modified nucleosides into extracellular fluids such as serum and urine. Here we searched for COVID-19-specific changes in modi
Externí odkaz:
https://doaj.org/article/fd140ee4687e4b7fbff56f17a96badec
Autor:
Eriko Yamaguchi, Kazuhiro Yoshikawa, Izaya Nakaya, Karen Kato, Yoshikazu Miyasato, Terumasa Nakagawa, Yutaka Kakizoe, Masashi Mukoyama, Jun Soma
Publikováno v:
BMC Nephrology, Vol 19, Iss 1, Pp 1-5 (2018)
Abstract Background Liddle’s syndrome is a rare monogenic form of hypertension caused by truncating or missense mutations in the C termini of the epithelial sodium channel (ENaC) β or γ subunits. Patients with this syndrome present with early ons
Externí odkaz:
https://doaj.org/article/f2a46d3991224a3e8ecfa613815c7858
Autor:
Yutaka Kakizoe, Yoshikazu Miyasato, Tomoaki Onoue, Terumasa Nakagawa, Manabu Hayata, Kohei Uchimura, Jun Morinaga, Teruhiko Mizumoto, Masataka Adachi, Taku Miyoshi, Yoshiki Sakai, Kimio Tomita, Masashi Mukoyama, Kenichiro Kitamura
Publikováno v:
Journal of Pharmacological Sciences, Vol 132, Iss 2, Pp 145-153 (2016)
Emerging evidence has suggested that aldosterone has direct deleterious effects on the kidney independently of its hemodynamic effects. However, the detailed mechanisms of these direct effects remain to be elucidated. We have previously reported that
Externí odkaz:
https://doaj.org/article/4f9c720a207e42d89d856d7808a43e91
Autor:
Yuki Narita, Miki Ueda, Kohei Uchimura, Yutaka Kakizoe, Yoshikazu Miyasato, Teruhiko Mizumoto, Jun Morinaga, Manabu Hayata, Terumasa Nakagawa, Masataka Adachi, Taku Miyoshi, Yoshiki Sakai, Daisuke Kadowaki, Sumio Hirata, Masashi Mukoyama, Kenichiro Kitamura
Publikováno v:
Journal of Pharmacological Sciences, Vol 130, Iss 2, Pp 110-116 (2016)
We previously reported that camostat mesilate (CM) had renoprotective and antihypertensive effects in rat CKD models. In this study, we examined if CM has a distinct renoprotective effect from telmisartan (TE), a renin-angiotensin-aldosterone system
Externí odkaz:
https://doaj.org/article/a0568c95b4384664a6769932401155f7