Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Yusur Falah Faraj"'
Publikováno v:
Mustansiriya Medical Journal, Vol 23, Iss 1, Pp 38-44 (2024)
Background: Despite the promising of introduction of tyrosine kinase inhibitors (TKIs), chronic myeloid leukemia (CML) remains a significant cause of annual mortality. Red blood cell distribution width (RDW), neutrophil/lymphocyte ratio (NLR), and pl
Externí odkaz:
https://doaj.org/article/2ada09bd47874fe7bf1bd8bfbd3ecfb9
Publikováno v:
Iraqi Journal of Hematology, Vol 13, Iss 1, Pp 154-156 (2024)
Externí odkaz:
https://doaj.org/article/d35b5aec1faa4f57a05e5cc01ddf6f43
Publikováno v:
Iraqi Journal of Hematology, Vol 12, Iss 2, Pp 184-189 (2023)
BACKGROUND: As in other malignancies, different subsets of natural killer (NK) cells play a crucial role in the recognition and lysing of malignant cells in chronic myeloid leukemia (CML). OBJECTIVES: This study aims to identify two subsets of NK, cy
Externí odkaz:
https://doaj.org/article/de9c1dc4a32149efbdd08e096ff523db
Publikováno v:
AIP Conference Proceedings.
Autor:
Jamela Jouda, Yusur Falah Faraj, Shatha Salah Asad, Ali Sameer Abudlghani Altaee, Khalid Mahdi Salih, Iyden Kamil Mohammed
Publikováno v:
Journal of Contemporary Medical Sciences. 5:90-95
Objectives: It is well established that diet and lifestyle are important in maintenance of healthy. Transition from a plant-based diet mostly to a high-calorie diet of animal products might raise the chronic diseases which called “degenerative
Publikováno v:
2ND INTERNATIONAL CONFERENCE ON MATERIALS ENGINEERING & SCIENCE (IConMEAS 2019).
Our previous work showed that changing in nutritional style play an important role in modifying the architectural aspects of various organs at the tissue level such as intestine and liver. Those findings need further investigation at physiological le
Publikováno v:
Iraqi Journal of Hematology, Vol 9, Iss 2, Pp 138-144 (2020)
BACKGROUND: Chronic immune thrombocytopenic purpura (ITP) is an immune-mediated bleeding disorder, in which platelets are opsonized by autoantibodies directed against platelet surface membrane glycoproteins, and prematurely cleared and destructed by