Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Yusuke Mieda"'
Autor:
Tomokazu Matsuda, Hiroaki Takahashi, Yusuke Mieda, Shinobu Shimizu, Takeshi Kawamoto, Yuki Matsuura, Tomoko Takai, Emi Suzuki, Ayumi Kanno, Maki Koyanagi-Kimura, Shun-ichiro Asahara, Alberto Bartolome, Norihide Yokoi, Hiroshi Inoue, Wataru Ogawa, Susumu Seino, Yoshiaki Kido
Publikováno v:
PLoS ONE, Vol 10, Iss 6, p e0130757 (2015)
During the development of type 2 diabetes, endoplasmic reticulum (ER) stress leads to not only insulin resistance but also to pancreatic beta cell failure. Conversely, cell function under various stressed conditions can be restored by reducing ER str
Externí odkaz:
https://doaj.org/article/8b5e6be2e4a64e57bed35e4a3c13a23d
Autor:
Emi, Suzuki, Tomokazu, Matsuda, Takeshi, Kawamoto, Hiroaki, Takahashi, Yusuke, Mieda, Yuki, Matsuura, Tomoko, Takai, Ayumi, Kanno, Maki, Koyanagi-Kimura, Shun-Ichiro, Asahara, Hiroshi, Inoue, Wataru, Ogawa, Yoshiaki, Kido
Publikováno v:
The Kobe journal of medical sciences. 64(2)
Endoplasmic reticulum (ER) stress leads to peripheral insulin resistance and the progression of pancreatic beta cell failure in type 2 diabetes. Although ER stress plays an important role in the pathogenesis of diabetes, it is indispensable for cellu
Autor:
Tomoko Takai, Shun-ichiro Asahara, Norihide Yokoi, Maki Koyanagi-Kimura, Emi Suzuki, Yoshiaki Kido, Ayumi Kanno, Hiroaki Takahashi, Yuki Matsuura, Tomokazu Matsuda, Yusuke Mieda, Wataru Ogawa, Hiroshi Inoue, Susumu Seino, Shinobu Shimizu, Alberto Bartolomé, Takeshi Kawamoto
Publikováno v:
PLoS ONE
PLoS ONE, Vol 10, Iss 6, p e0130757 (2015)
PLoS ONE, Vol 10, Iss 6, p e0130757 (2015)
During the development of type 2 diabetes, endoplasmic reticulum (ER) stress leads to not only insulin resistance but also to pancreatic beta cell failure. Conversely, cell function under various stressed conditions can be restored by reducing ER str
Autor:
Hiroaki Etoh, Ayumi Kanno, Shinobu Shimizu, Hiroaki Takahashi, Yusuke Mieda, Hiroyuki Inoue, Yoshiaki Kido, Susumu Seino, Maki Koyanagi-Kimura, Alberto Bartolomé, Shun-ichiro Asahara, Kyoko Teruyama, Megumi Fuchita, Naoko Hashimoto, Tetsuya Hosooka, Tomokazu Matsuda
Publikováno v:
Journal of molecular endocrinology. 49(2)
The development of type 2 diabetes is accompanied by a progressive decline in β-cell mass and function. Vildagliptin, a dipeptidyl peptidase 4 inhibitor, is representative of a new class of antidiabetic agents that act through increasing the express