Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Yurie Sato-Yamada"'
Autor:
Kridtapat Sirisereephap, Hikaru Tamura, Jong-Hyung Lim, Meircurius Dwi Condro Surboyo, Toshihito Isono, Takumi Hiyoshi, Andrea L. Rosenkranz, Yurie Sato-Yamada, Hisanori Domon, Akari Ikeda, Tomoyasu Hirose, Toshiaki Sunazuka, Nagako Yoshiba, Hiroyuki Okada, Yutaka Terao, Takeyasu Maeda, Koichi Tabeta, Triantafyllos Chavakis, George Hajishengallis, Tomoki Maekawa
Publikováno v:
iScience, Vol 27, Iss 2, Pp 108798- (2024)
Summary: Aging is associated with increased susceptibility to chronic inflammatory bone loss disorders, such as periodontitis, in large part due to the impaired regenerative potential of aging tissues. DEL-1 exerts osteogenic activity and promotes bo
Externí odkaz:
https://doaj.org/article/b4d1b2b7ec0e4334957f28a8a3880467
Autor:
Yurie Sato-Yamada, Amy Strickland, Yo Sasaki, Joseph Bloom, Aaron DiAntonio, Jeffrey Milbrandt
Publikováno v:
The Journal of Clinical Investigation, Vol 132, Iss 23 (2022)
Charcot-Marie-Tooth disease type 2A (CMT2A) is an axonal neuropathy caused by mutations in the mitofusin 2 (MFN2) gene. MFN2 mutations result in profound mitochondrial abnormalities, but the mechanism underlying the axonal pathology is unknown. Steri
Externí odkaz:
https://doaj.org/article/a22016f1de27458fb151f9e0cd2dd534
Autor:
Yurie Sato-Yamada, Amy Strickland, Yo Sasaki, Joseph Bloom, Aaron DiAntonio, Jeffrey Milbrandt
Charcot-Marie-Tooth disease (CMT) type 2A is an axonal neuropathy caused by mutations in the mitofusin 2 (MFN2) gene. MFN2 mutations result in profound mitochondrial abnormalities, but the mechanism underlying axonal pathology is unknown. SARM1, the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::8c09ef29321c303cfae5271badabc612
https://doi.org/10.1101/2022.05.17.492364
https://doi.org/10.1101/2022.05.17.492364