Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Yurie Hasegawa"'
Autor:
Yurie Hasegawa, Yohei Iwata, Hidehiko Fukushima, Yoshihito Tanaka, Soichiro Watanabe, Kenta Saito, Hiroyuki Ito, Mizuki Sugiura, Masashi Akiyama, Kazumitsu Sugiura
Publikováno v:
Scientific Reports, Vol 12, Iss 1, Pp 1-9 (2022)
Abstract Loss-of-function homozygous or compound heterozygous mutations in IL36RN, which encodes interleukin-36 receptor antagonist (IL-36Ra), have been implicated in the pathogenesis of skin disorders. We previously reported that Il36rn −/− mice
Externí odkaz:
https://doaj.org/article/32411d106da94441868e51f1c7445b5e
Autor:
Kenta Saito, Hidehiko Fukushima, Yohei Iwata, Masashi Akiyama, Soichiro Watanabe, Yoshihito Tanaka, Yurie Hasegawa, Kazumitsu Sugiura
Publikováno v:
Journal of the European Academy of Dermatology and Venereology. 36:295-304
BACKGROUND Loss-of-function homozygous or compound heterozygous mutations in IL36RN, which encodes interleukin-36 receptor antagonist (IL-36Ra), has been implicated in the pathogenesis of skin disorders. However, the pathogenic role of IL-36Ra in cut
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::1b14fcc0899ec8eefbf8b09a1518bc55
https://doi.org/10.1111/jdv.17767
https://doi.org/10.1111/jdv.17767
Autor:
Soichiro Watanabe, Yoshihito Tanaka, Yurie Hasegawa, Hidehiko Fukushima, Kazumitsu Sugiura, Kenta Saito, Masashi Akiyama, Yohei Iwata
Publikováno v:
Scientific Reports
Scientific Reports, Vol 10, Iss 1, Pp 1-12 (2020)
Scientific Reports, Vol 10, Iss 1, Pp 1-12 (2020)
Loss-of-function homozygous or compound heterozygous mutations in IL36RN, which encodes interleukin-36 receptor antagonist (IL-36Ra), have been implicated in the pathogenesis of various skin disorders. Previous findings showed that IL-36γ promoted w
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7eca065d885a4d21e3d165f373783b5d
http://europepmc.org/articles/PMC7479622
http://europepmc.org/articles/PMC7479622
Autor:
Yoshihito Tanaka, Yurie Hasegawa, Soichiro Watanabe, Yohei Iwata, Hidehiko Fukushima, Masashi Akiyama, Kazumitsu Sugiura, Kenta Saito
Publikováno v:
Scientific Reports
Scientific Reports, Vol 10, Iss 1, Pp 1-11 (2020)
Scientific Reports, Vol 10, Iss 1, Pp 1-11 (2020)
Loss-of-function mutations in the interleukin (IL)-36 gene IL36RN are associated with psoriasis. The importance of neutrophil extracellular traps (NETs), web-like structures composed of neutrophil DNA, in the pathogenesis of psoriasis has been unclea
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::59f8318ab64a4897d0927265d22ccdf6
https://pubmed.ncbi.nlm.nih.gov/33214582
https://pubmed.ncbi.nlm.nih.gov/33214582
Autor:
Yoshihito Tanaka, Yurie Hasegawa, Masashi Akiyama, Kazumitsu Sugiura, Yohei Iwata, Kenta Saito, Soichiro Watanabe, Hidehiko Fukushima
Publikováno v:
Scientific Reports
Scientific Reports, Vol 10, Iss 1, Pp 1-10 (2020)
Scientific Reports, Vol 10, Iss 1, Pp 1-10 (2020)
Loss-of-function mutations in IL36RN cause generalized pustular psoriasis (GPP), which is characterized by neutrophil-infiltrated lesions. Neutrophils are important during contact hypersensitivity in mice. However, it has never been determined whethe
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::53a3db83082199e3893818daeb0ddcb7
http://europepmc.org/articles/PMC6971010
http://europepmc.org/articles/PMC6971010