Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Yuri Kasagi"'
Autor:
Yuki Yoshizaki, Takayasu Mori, Mari Ishigami-Yuasa, Eriko Kikuchi, Daiei Takahashi, Moko Zeniya, Naohiro Nomura, Yutaro Mori, Yuya Araki, Fumiaki Ando, Shintaro Mandai, Yuri Kasagi, Yohei Arai, Emi Sasaki, Sayaka Yoshida, Hiroyuki Kagechika, Tatemitsu Rai, Shinichi Uchida, Eisei Sohara
Publikováno v:
Scientific Reports, Vol 7, Iss 1, Pp 1-10 (2017)
Abstract The Kelch-like ECH-associating protein 1 (Keap1)-nuclear factor erythroid 2-related factor 2 (Nrf2)-antioxidant response element (ARE) signaling pathway is the major regulator of cytoprotective responses to oxidative and electrophilic stress
Externí odkaz:
https://doaj.org/article/9033bda222a94a88a5557b6bef9e8da0
Autor:
Yuya Araki, Eisei Sohara, Hiroyuki Kagechika, Eriko Kikuchi, Daiei Takahashi, Emi Sasaki, Yuki Yoshizaki, Naohiro Nomura, Mari Ishigami-Yuasa, Takayasu Mori, Sayaka Yoshida, Moko Zeniya, Tatemitsu Rai, Fumiaki Ando, Shinichi Uchida, Yohei Arai, Yutaro Mori, Shintaro Mandai, Yuri Kasagi
Publikováno v:
Scientific Reports, Vol 7, Iss 1, Pp 1-10 (2017)
Scientific Reports
Scientific Reports
The Kelch-like ECH-associating protein 1 (Keap1)-nuclear factor erythroid 2-related factor 2 (Nrf2)-antioxidant response element (ARE) signaling pathway is the major regulator of cytoprotective responses to oxidative and electrophilic stress. The Cul
Autor:
Eisei Sohara, Kiyoshi Isobe, Tomokazu Okado, Yuya Araki, Yuri Kasagi, Pascale Bomont, Koichiro Susa, Yuichi Inoue, Emi Sasaki, Tatemitsu Rai, Sayaka Yoshida, Shinichi Uchida, Takayasu Mori
Publikováno v:
Clinical and experimental nephrology. 22(6)
Pseudohypoaldosteronism type II (PHAII) is a hereditary hypertensive disease caused by mutations in four genes: WNK1, WNK4, Kelch-like3 (KLHL3), and cullin3 (CUL3). Recently, it was revealed that CUL3–KLHL3 E3 ligase complex ubiquitinates WNK1 and
Autor:
Tatemitsu Rai, Tomomi Aida, Eisei Sohara, Shinichi Uchida, Hidenori Nishida, Daiei Takahashi, Fumiaki Ando, Moko Zeniya, Naohiro Nomura, Takayasu Mori, Emi Sasaki, Yuri Kasagi
Publikováno v:
Biochemical and biophysical research communications. 487(2)
Mutations in the with-no-lysine kinase 1 (WNK1), WNK4, Kelch-like 3 (KLHL3), and Cullin3 (CUL3) genes were identified as being responsible for hereditary hypertensive disease pseudohypoaldosteronism type II (PHAII). Normally, the KLHL3/CUL3 ubiquitin
Autor:
Shinichi Uchida, Yohei Arai, Shintaro Mandai, Fumiaki Ando, Daiei Takahashi, Yuri Kasagi, Sayaka Yoshida, Naohiro Nomura, Takayasu Mori, Yasuro Furuichi, Yutaka Hata, Emi Sasaki, Susumu Furukawa, Manami Kodaka, Eisei Sohara, Tatemitsu Rai, Yuki Yoshizaki, Yutaro Mori, Nobuharu L. Fujii
Publikováno v:
Scientific Reports
Muscle wasting or sarcopenia contributes to morbidity and mortality in patients with cancer, renal failure, or heart failure, and in elderly individuals. Na+-K+-2Cl− cotransporter 1 (NKCC1) is highly expressed in mammalian skeletal muscle, where it
Publikováno v:
Journal of infection and chemotherapy : official journal of the Japan Society of Chemotherapy. 20(11)
A 48-year-old man with autosomal dominant polycystic kidney disease (ADPKD) was admitted to our hospital with a 5-day history of lower right back pain, high-grade fever, and arthralgia. He was diagnosed with right kidney cyst infection and bacteremia
Autor:
Satomi Shikuma, Michio Kuwahara, Wataru Akita, Tomomi Tanaka, Yuri Kasagi, Shintaro Mandai, Keita Kusaka
Publikováno v:
Clinical and experimental nephrology. 19(4)
Renal anemia of chronic kidney disease (CKD) is generally treated by erythropoiesis-stimulating agents (ESAs). However, there are individual differences in patients’ responsiveness to ESA, which may affect the prognosis of CKD. The effect of ESAs o
Autor:
Hiroaki Yokote, Hiroto Fujigasaki, Masahiko Ichijo, Mitsuaki Sato, Hirotomo Miake, Yuri Kasagi
Publikováno v:
Nihon Naika Gakkai Zasshi. 98:3146-3147
Autor:
Yuri Kasagi, Tomomi Tanaka, Sei Sasaki, Keita Kusaka, Shintaro Mandai, Michio Kuwahara, Wataru Akita, Satomi Shikuma
Publikováno v:
BMC Nephrology
Background Hyponatremia is associated with increased mortality in chronic kidney disease with and without end-stage renal disease (ESRD). Increasing evidence suggests that hyponatremia is not only a marker of severe underlying disease, but also a dir