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Publikováno v:
Frontiers in Cellular Neuroscience, Vol 8 (2014)
One of the most critical pathological features of Alzheimer’s disease (AD) is the accumulation of β-amyloid (Aβ) peptides that form extracellular senile plaques in the brain. Aβ is derived from β-amyloid precursor protein through sequential cle
Externí odkaz:
https://doaj.org/article/4a0d77446a0f43aab11b359bb8c523a1
