Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Yumiko M. Araki"'
Autor:
Wataru Araki, Kotaro Hattori, Kazutomi Kanemaru, Yuma Yokoi, Yoshie Omachi, Harumasa Takano, Masuhiro Sakata, Sumiko Yoshida, Tadashi Tsukamoto, Miho Murata, Yuko Saito, Hiroshi Kunugi, Yu-ichi Goto, Utako Nagaoka, Masahiro Nagao, Takashi Komori, Kunimasa Arima, Kenji Ishii, Shigeo Murayama, Hiroshi Matsuda, Hisateru Tachimori, Yumiko M. Araki, Hidehiro Mizusawa
Publikováno v:
Biomarker Research, Vol 5, Iss 1, Pp 1-9 (2017)
Abstract Background Because soluble (or secreted) amyloid precursor protein-β (sAPPβ) and -α (sAPPα) possibly reflect pathological features of Alzheimer’s disease (AD), they are potential biomarker candidates for dementia disorders, including A
Externí odkaz:
https://doaj.org/article/38a7784e74c34cf2a2ddcd352fc4a090
Publikováno v:
Neurology and Clinical Neuroscience. 6:89-93
Autor:
Kazutomi Kanemaru, Kenji Ishii, Yuko Saito, Masuhiro Sakata, Kotaro Hattori, Wataru Araki, Masahiro Nagao, Yoshie Omachi, Yuma Yokoi, Hidehiro Mizusawa, Hiroshi Matsuda, Yumiko M. Araki, Sumiko Yoshida, Shigeo Murayama, Yu-ichi Goto, Harumasa Takano, Utako Nagaoka, Hiroshi Kunugi, Hisateru Tachimori, Tadashi Tsukamoto, Kunimasa Arima, Takashi Komori, Miho Murata
Publikováno v:
Biomarker Research, Vol 5, Iss 1, Pp 1-9 (2017)
Biomarker Research
Biomarker Research
Background Because soluble (or secreted) amyloid precursor protein-β (sAPPβ) and -α (sAPPα) possibly reflect pathological features of Alzheimer’s disease (AD), they are potential biomarker candidates for dementia disorders, including AD and mil
Autor:
Akira Tamaoka, Akiyoshi Saitoh, Kaori Taniguchi, Daisuke Yamada, Masayuki Sekiguchi, Wataru Araki, Hidehiro Mizusawa, Fumiko Yamamoto, Jinwei Yang, Fuyuki Kametani, Yusuke Sakai, Keiji Wada, Yumiko M. Araki, Naomi Mamada, Takuya Arai, Masayuki Itoh
Publikováno v:
Journal of Alzheimer's disease : JAD. 70(3)
Soluble amyloid-β (Aβ) oligomers (AβOs), which elicit neurotoxicity and synaptotoxicity, are thought to play an initiating role in the pathology of Alzheimer's disease (AD). Since AβOs are a key therapeutic target, we attempted to identify natura
Autor:
Hideaki Kume, Hirohiko Hohjoh, Wataru Araki, Yumiko M. Araki, Kazumi Motoki, Akira Tamaoka, Seiji Minegishi
Publikováno v:
Molecular neurobiology. 50(3)
TAR DNA-binding protein (TDP-43) is a major component of most ubiquitin-positive neuronal and glial inclusions of amyotrophic lateral sclerosis (ALS) and frontotemporal lobar degeneration (FTLD). A number of missense mutations in the TARDBP gene have