Zobrazeno 1 - 10
of 158
pro vyhledávání: '"Yuko Miyagoe-Suzuki"'
Autor:
Fusako Sakai‐Takemura, Fumiaki Saito, Ken'ichiro Nogami, Yusuke Maruyama, Ahmed Elhussieny, Kiichiro Matsumura, Shin'ichi Takeda, Yoshitsugu Aoki, Yuko Miyagoe‐Suzuki
Publikováno v:
FASEB BioAdvances, Vol 5, Iss 11, Pp 453-469 (2023)
Abstract Store‐operated Ca2+ entry (SOCE) is indispensable for intracellular Ca2+ homeostasis in skeletal muscle, and constitutive activation of SOCE causes tubular aggregate myopathy (TAM). To understand the pathogenesis of TAM, we induced pluripo
Externí odkaz:
https://doaj.org/article/9ab6fb4209e344dcab30a52c6893f7f7
Autor:
Ahmed Elhussieny, Ken’ichiro Nogami, Fusako Sakai-Takemura, Yusuke Maruyama, Natsumi Takemura, Wael Talaat Soliman, Shin’ichi Takeda, Yuko Miyagoe-Suzuki
Publikováno v:
Stem Cell Research & Therapy, Vol 12, Iss 1, Pp 1-16 (2021)
Abstract Background Duchenne muscular dystrophy (DMD) is a severe X-linked recessive disease caused by mutations in the dystrophin gene. Transplantation of myogenic stem cells holds great promise for treating muscular dystrophies. However, poor engra
Externí odkaz:
https://doaj.org/article/b33ad373fd264489862b6acd610311d8
Autor:
Ken’ichiro Nogami, Yusuke Maruyama, Ahmed Elhussieny, Fusako Sakai-Takemura, Jun Tanihata, Jun-ichi Kira, Yuko Miyagoe-Suzuki, Shin’ichi Takeda
Publikováno v:
BMC Musculoskeletal Disorders, Vol 21, Iss 1, Pp 1-10 (2020)
Abstract Background Previous research indicated that nitric oxide synthase (NOS) is the key molecule for S-nitrosylation of ryanodine receptor 1 (RyR1) in DMD model mice (mdx mice) and that both neuronal NOS (nNOS) and inducible NOS (iNOS) might cont
Externí odkaz:
https://doaj.org/article/75d1c8790bdd41cabdfac1b9a7743377
Autor:
Shigefumi Morioka, Hirofumi Sakaguchi, Hiroaki Mohri, Mariko Taniguchi-Ikeda, Motoi Kanagawa, Toshiaki Suzuki, Yuko Miyagoe-Suzuki, Tatsushi Toda, Naoaki Saito, Takehiko Ueyama
Publikováno v:
PLoS Genetics, Vol 16, Iss 5, p e1008826 (2020)
Hearing loss (HL) is one of the most common sensory impairments and etiologically and genetically heterogeneous disorders in humans. Muscular dystrophies (MDs) are neuromuscular disorders characterized by progressive degeneration of skeletal muscle a
Externí odkaz:
https://doaj.org/article/e459f0bf0deb4f4bba4537cab949d556
Publikováno v:
International Journal of Molecular Sciences, Vol 22, Iss 7, p 3627 (2021)
Skeletal muscle regeneration is a well-organized process that requires remodeling of the extracellular matrix (ECM). In this study, we revealed the protective role of periostin, a matricellular protein that binds to several ECM proteins during muscle
Externí odkaz:
https://doaj.org/article/ead8c05410724ed587290a0129016c5f
Autor:
Akiyoshi Uezumi, Masashi Nakatani, Madoka Ikemoto-Uezumi, Naoki Yamamoto, Mitsuhiro Morita, Asami Yamaguchi, Harumoto Yamada, Takehiro Kasai, Satoru Masuda, Asako Narita, Yuko Miyagoe-Suzuki, Shin’ichi Takeda, So-ichiro Fukada, Ichizo Nishino, Kunihiro Tsuchida
Publikováno v:
Stem Cell Reports, Vol 7, Iss 2, Pp 263-278 (2016)
Skeletal muscle contains two distinct stem/progenitor populations. One is the satellite cell, which acts as a muscle stem cell, and the other is the mesenchymal progenitor, which contributes to muscle pathogeneses such as fat infiltration and fibrosi
Externí odkaz:
https://doaj.org/article/1ed32a83306f4a04a97862709baae44e
Publikováno v:
Cells, Vol 9, Iss 11, p 2524 (2020)
Perlecan is an extracellular matrix molecule anchored to the sarcolemma by a dystrophin–glycoprotein complex. Perlecan-deficient mice are tolerant to muscle atrophy, suggesting that perlecan negatively regulates mechanical stress-dependent skeletal
Externí odkaz:
https://doaj.org/article/5ff16f0a0b4240fe9c0857b81e375a67
Autor:
Masahiko Yamaguchi, Yoko Watanabe, Takuji Ohtani, Akiyoshi Uezumi, Norihisa Mikami, Miki Nakamura, Takahiko Sato, Masahito Ikawa, Mikio Hoshino, Kunihiro Tsuchida, Yuko Miyagoe-Suzuki, Kazutake Tsujikawa, Shin’ichi Takeda, Hiroshi Yamamoto, So-ichiro Fukada
Publikováno v:
Cell Reports, Vol 13, Iss 2, Pp 302-314 (2015)
Calcitonin receptor (Calcr) is expressed in adult muscle stem cells (muscle satellite cells [MuSCs]). To elucidate the role of Calcr, we conditionally depleted Calcr from adult MuSCs and found that impaired regeneration after muscle injury correlated
Externí odkaz:
https://doaj.org/article/5d5c7ed7ec964b9b967d8575b02f9c47
Autor:
Yuka Watanabe, Yuko Miyagoe-Suzuki
Publikováno v:
Journal of Physical Fitness and Sports Medicine, Vol 4, Iss 1, Pp 73-82 (2015)
Muscle mass and strength decline with age. When severe, the loss is called sarcopenia. Sarcopenia is drawing attention worldwide, especially in highly aged societies, as a disease that should be treated. At present, we have limited tools to combat sa
Externí odkaz:
https://doaj.org/article/ceff07944ddb4d16866f5e08afddb054
Autor:
Yuko Miyagoe-Suzuki, Takashi Nishiyama, Miho Nakamura, Asako Narita, Fusako Takemura, Satoru Masuda, Narihiro Minami, Kumiko Murayama, Hirofumi Komaki, Yu-ichi Goto, Shin’ichi Takeda
Publikováno v:
Stem Cells International, Vol 2017 (2017)
Three to eight percent of female carriers of Duchenne muscular dystrophy (DMD) develop dystrophic symptoms ranging from mild muscle weakness to a rapidly progressive DMD-like muscular dystrophy due to skewed inactivation of X chromosomes during early
Externí odkaz:
https://doaj.org/article/ffeaf6e9c17b4ba8921ef92953d5cd2a