Zobrazeno 1 - 10
of 90
pro vyhledávání: '"Yukio Nishizawa"'
Publikováno v:
Journal of Pharmacological Sciences, Vol 108, Iss 4, Pp 517-528 (2008)
K+ channels are key modulators of neuronal excitability, and mutations in certain types of these channels are known to cause epileptic seizures. Activation of K+ channels is reported to suppress epileptic discharge; however, the types of K+-channel o
Externí odkaz:
https://doaj.org/article/69dfe8f11cc044518599e7c35f883699
Autor:
Kazunori Kaede, Toshihiro Shiimado, Masanori Matsuoka, Yukio Nishizawa, Keiichi Watanuki, Keiichi Muramatsu
Publikováno v:
SAE Technical Paper Series.
Publikováno v:
SAE Technical Paper Series.
Publikováno v:
SAE International Journal of Commercial Vehicles. 7:703-709
Publikováno v:
TRANSACTIONS OF THE JAPAN SOCIETY OF MECHANICAL ENGINEERS Series C. 78:382-395
Preventing brake squeal is a significant task in brake development because brake squeal bothers users, thereby reducing a vehicle's commercial value. Many researchers have tried to find the mechanisms that cause brake squeal, so the number of complai
Autor:
Yukio Nishizawa, Yusuke Aoki, Yutaka Kurita, Yasunori Oura, Takashi Tanaka, Masaki Kanemoto, Yasuyuki Kanehira, Satoshi Obata, Kazuma Watanabe
Publikováno v:
The Proceedings of Mechanical Engineering Congress, Japan. 2018:G1000505
Publikováno v:
Journal of System Design and Dynamics. 4:970-983
A squeal test using a pad with a different thickness demonstrated that a squeal with a higher frequency can be generated if a thin pad is used. The factors that changed as a result of pad thickness were termed 'difference of pad rigidity' and 'dimens
Publikováno v:
Transactions of the Japan Society of Mechanical Engineers Series C. 76:2552-2559
Publikováno v:
Transactions of the Japan Society of Mechanical Engineers Series C. 76:1287-1294
Autor:
Rosamund Dove, Lechoslaw Turski, Alim Uddin, Bela Patel, Terence Smith, Jia Newcombe, Yukio Nishizawa
Publikováno v:
Brain Pathol
Blockade of receptors for the excitatory neurotransmitter glutamate ameliorates neurological clinical signs in models of the CNS inflammatory demyelinating disease multiple sclerosis (MS). To investigate whether glutamate excitoxicity may play a role