Zobrazeno 1 - 10
of 31
pro vyhledávání: '"Yukio Kim"'
Publikováno v:
Scientific Reports, Vol 14, Iss 1, Pp 1-9 (2024)
Abstract Abnormal brain aging is suggested in epilepsy. Given the brain network dysfunction in epilepsy, the white matter tracts, which primarily interconnect brain regions, could be of special importance. We focused on white matter brain aging in di
Externí odkaz:
https://doaj.org/article/eedf339d692d402a8db4b5a78edd2148
Autor:
Keiya Iijima, Hiroyuki Fujii, Fumio Suzuki, Kumiko Murayama, Yu-ichi Goto, Yuko Saito, Terunori Sano, Hiroyoshi Suzuki, Hajime Miyata, Yukio Kimura, Takuma Nakashima, Hiromichi Suzuki, Masaki Iwasaki, Noriko Sato
Publikováno v:
Frontiers in Neurology, Vol 15 (2024)
IntroductionLow-grade epilepsy-associated tumors are the second most common histopathological diagnoses in cases of drug-resistant focal epilepsy. However, the connection between neuroimaging features and genetic alterations in these tumors is unclea
Externí odkaz:
https://doaj.org/article/ffdfde74e10e4215bbcbe99475285cd0
Autor:
Sy Le Thanh Nguyen, Thi Hien Trang Nguyen, Thi Tuyen Do, Thi Thao Nguyen, Thanh Hoang Le, Thi Anh Tuyet Nguyen, Yukio Kimata
Publikováno v:
Current Issues in Molecular Biology, Vol 46, Iss 3, Pp 1768-1776 (2024)
Prodigiosin, a red pigment produced by numerous bacterial species, exerts various antibiotic effects on prokaryotic and eukaryotic organisms. For instance, human carcinoma cell lines appear to suffer from endoplasmic reticulum (ER) stress in the pres
Externí odkaz:
https://doaj.org/article/a8c57ae408254034963239601560bcb1
Publikováno v:
Brain Sciences, Vol 14, Iss 10, p 992 (2024)
Background/Objectives: Although the involvement of progressive brain alterations in epilepsy was recently suggested, individual patients’ trajectories of white matter (WM) disruption are not known. Methods: We investigated the disease progression p
Externí odkaz:
https://doaj.org/article/bb439d19ca554463a09b2606e36f5d42
Autor:
Ashuei Sogawa, Ryota Komori, Kota Yanagitani, Miku Ohfurudono, Akio Tsuru, Koji Kadoi, Yukio Kimata, Hiderou Yoshida, Kenji Kohno
Publikováno v:
Cell Structure and Function, Vol 48, Iss 2, Pp 211-221 (2023)
Secretory pathway proteins are cotranslationally translocated into the endoplasmic reticulum (ER) of metazoan cells through the protein channel, translocon. Given that there are far fewer translocons than ribosomes in a cell, it is essential that sec
Externí odkaz:
https://doaj.org/article/cad2245e20b043869e0ac315ead82998
Autor:
Elly Arizono, Zen-ichi Tanei, Keiya Iijima, Yukio Kimura, Yoko Shigemoto, Hiroyuki Maki, Midori Kusama, Kumiko Murayama, Masaki Iwasaki, Takashi Saito, Yuko Saito, Kazuhiro Saito, Noriko Sato
Publikováno v:
Epilepsy & Behavior Reports, Vol 26, Iss , Pp 100674- (2024)
Mild malformation of cortical development with oligodendroglial hyperplasia (MOGHE) is a recently proposed epileptogenic entity that is difficult to detect on MRI. We present a case of MOGHE that was successfully detected on T1WI-chemical shift-selec
Externí odkaz:
https://doaj.org/article/53fce872d8704aff9a5d1cbb024b27e9
Publikováno v:
Epilepsia Open, Vol 7, Iss 4, Pp 657-664 (2022)
Abstract Objective Psychosis is an important comorbidity in epilepsy, but its pathophysiology is still unknown. The imaging modality 18F‐fluorodeoxyglucose‐positron emission tomography (18F‐FDG PET) is widely used to measure brain glucose metab
Externí odkaz:
https://doaj.org/article/f0c7228562cc44369d68d02e2a4e6af7
Publikováno v:
Frontiers in Microbiology, Vol 14 (2023)
In eukaryotic species, dysfunction of the endoplasmic reticulum (ER), namely, ER stress, provokes a cytoprotective transcription program called the unfolded protein response (UPR). The UPR is triggered by transmembrane ER-stress sensors, including Ir
Externí odkaz:
https://doaj.org/article/95eae72ded344200bac56084aa67fc16
Autor:
Yuki Ishiwata-Kimata, Yukio Kimata
Publikováno v:
Journal of Fungi, Vol 9, Iss 10, p 989 (2023)
Upon the dysfunction or functional shortage of the endoplasmic reticulum (ER), namely, ER stress, eukaryotic cells commonly provoke a protective gene expression program called the unfolded protein response (UPR). The molecular mechanism of UPR has be
Externí odkaz:
https://doaj.org/article/a20198e89da6460e92fd12e433fe6b88
Autor:
Wakiro Sato, Daisuke Noto, Manabu Araki, Tomoko Okamoto, Youwei Lin, Hiromi Yamaguchi, Ryoko Kadowaki-Saga, Atsuko Kimura, Yukio Kimura, Noriko Sato, Takami Ishizuka, Harumasa Nakamura, Sachiko Miyake, Takashi Yamamura
Publikováno v:
Therapeutic Advances in Neurological Disorders, Vol 16 (2023)
Background: Multiple sclerosis (MS) is an autoimmune inflammatory disease of the central nervous system that causes the damage to the myelin sheath as well as axonal degeneration. Individuals with MS appear to have changes in the numbers and function
Externí odkaz:
https://doaj.org/article/2ec57a84f74c469d8c2030fb36813dfd