Zobrazeno 1 - 10
of 13
pro vyhledávání: '"Yuka Katsumata"'
Autor:
Takayuki Kunisato, Mikio Watanabe, Naoya Inoue, Azusa Okada, Takashi Nanba, Wataru Kobayashi, Yuka Inoue, Yuka Katsumata, Naoki Omori, Takayuki Nobuhara, Kazuya Takemura, Yoh Hidaka, Yoshinori Iwatani
Publikováno v:
Autoimmunity, Vol 51, Iss 7, Pp 360-369 (2018)
The prognosis of autoimmune thyroid disease (AITD) including Graves’ disease (GD) and Hashimoto’s disease (HD) is difficult to predict. We previously suggested that Th17 cells may be associated with the pathogenesis of AITD. However, the associat
Externí odkaz:
https://doaj.org/article/a950b972b1024d25870173fac324059d
Autor:
Naoya Inoue, Yuka Katsumata, Mikio Watanabe, Naoko Ishido, Yu Manabe, Ayano Watanabe, Ryota Masutani, Yoh Hidaka, Yoshinori Iwatani
Publikováno v:
Autoimmunity, Vol 50, Iss 3, Pp 182-191 (2017)
Graves’ disease (GD) and Hashimoto’s disease (HD) are autoimmune thyroid diseases (AITDs). Prognosis of AITDs varies in each patient. Toll-like receptors (TLRs) are pattern-recognition receptors that activate signaling pathways involved in the pr
Externí odkaz:
https://doaj.org/article/42dab3f68763442cb68541970ce3db61
Autor:
Yukina Takuse, Yoh Hidaka, Naoya Inoue, Yuka Katsumata, Hiroshi Ohtsu, Minori Saeki, Mikio Watanabe, Yoshinori Iwatani, Ritsuko Ozaki
Publikováno v:
Immunological Investigations. 46:590-602
Interleukin (IL)-10 is known to suppress inflammation in autoimmune diseases. IL-10 can be regulated by miRNAs. To elucidate the involvement of miRNAs that regulate IL-10 expression with the pathogenesis of autoimmune thyroid disease (AITD), we exami
Autor:
Yoshinori Iwatani, Takayuki Nobuhara, Mikio Watanabe, Kazuya Takemura, Azusa Okada, Takashi Nanba, Yoh Hidaka, Yuka Katsumata, Takayuki Kunisato, Naoya Inoue, Naoki Omori, Wataru Kobayashi, Yuka Inoue
Publikováno v:
Autoimmunity. 51(7)
The prognosis of autoimmune thyroid disease (AITD) including Graves' disease (GD) and Hashimoto's disease (HD) is difficult to predict. We previously suggested that Th17 cells may be associated with the pathogenesis of AITD. However, the association
Publikováno v:
Immunological investigations. 47(5)
Graves' disease (GD) and Hashimoto's disease (HD) are major autoimmune thyroid diseases (AITDs), and their pathological conditions vary among patients. Type 1 iodothyronine deiodinase (D1) and type 2 iodothyronine deiodinase (D2) convert from thyroxi
Association between functional polymorphisms in the toll-like receptor 4 (TLR4) gene and HD severity
Publikováno v:
Tissue Antigens. 85:209-211
Autor:
Yoshinori Iwatani, Yu Manabe, Yuka Katsumata, Mikio Watanabe, Naoya Inoue, Ryota Masutani, Ayano Watanabe, Yoh Hidaka, Naoko Ishido
Publikováno v:
Autoimmunity. 50(3)
Graves' disease (GD) and Hashimoto's disease (HD) are autoimmune thyroid diseases (AITDs). Prognosis of AITDs varies in each patient. Toll-like receptors (TLRs) are pattern-recognition receptors that activate signaling pathways involved in the produc
Autor:
Mikio Watanabe, Yoshinori Iwatani, Yumi Miyahara, Yoh Hidaka, Maiko Akahane, Naoya Inoue, Yuka Katsumata, Yuya Arakawa, Yuka Inoue
Publikováno v:
Autoimmunity. 49(5)
Chemokines induce leukocyte chemotaxis and contribute to chronic inflammation. To clarify the association between functional polymorphisms in genes encoding some chemokines and the pathogenesis of Autoimmune thyroid disease (AITD), we genotyped IL8 -
Autor:
Mikio Watanabe, Minori Saeki, Yoshinori Iwatani, Hiroshi Otsu, Naoya Inoue, Yuka Katsumata, Yukina Takuse, Tomoya Kagawa
Publikováno v:
Endocrine journal. 63(4)
MicroRNA (miRNA) is a family of non-coding RNAs that have important roles in various vital functions. It has been reported that let-7e, a miRNA, may be involved in the regulation of interleukin (IL)-10 production. The purpose of this study was to eva
Autor:
Yuka Katsumata, Yoh Hidaka, Hayato Maruoka, Yoshinori Iwatani, Mikio Watanabe, Naoko Ishido, Arisa Kodu, Naoya Inoue
Publikováno v:
Human immunology. 77(10)
Apoptosis is necessary for the maintenance of self-tolerance by eliminating autoreactive immune cells in the periphery. To clarify the association between the pathogenesis of autoimmune thyroid disease (AITD) and genes encoding apoptosis regulatory f